Results 11 to 20 of about 9,866 (257)

Natriuresis‐guided therapy in acute heart failure: rationale and design of the Pragmatic Urinary Sodium‐based treatment algoritHm in Acute Heart Failure (PUSH‐AHF) trial

open access: yesEuropean Journal of Heart Failure, 2021
Insufficient diuretic response frequently occurs in patients admitted for acute heart failure (HF) and is associated with worse clinical outcomes. Recent studies have shown that measuring natriuresis early after hospital admission could reliably identify
J. T. ter Maaten   +8 more
semanticscholar   +1 more source

Acclimation to a High‐Salt Diet Is Sex Dependent

open access: yesJournal of the American Heart Association: Cardiovascular and Cerebrovascular Disease, 2022
Background Premenopausal women are less likely to develop hypertension and salt‐related complications than are men, yet the impact of sex on mechanisms regulating Na+ homeostasis during dietary salt challenges is poorly defined.
Eman Y. Gohar   +14 more
doaj   +1 more source

Renal sympathetic activity: A key modulator of pressure natriuresis in hypertension.

open access: yesBiochemical Pharmacology, 2022
Hypertension is a complex disorder ensuing necessarily from alterations in the pressure-natriuresis relationship, the main determinant of long-term control of blood pressure.
N. Díaz-Morales   +3 more
semanticscholar   +1 more source

Caffeine-induced diuresis and natriuresis is independent of renal tubular NHE3

open access: yesAmerican Journal of Physiology - Renal Physiology, 2015
Timo Rieg   +2 more
exaly   +2 more sources

Mineralocorticoid receptor antagonists cause natriuresis in the absence of aldosterone

open access: yesHYPERTENSION, 2022
Background: MR (mineralocorticoid receptor) antagonists are recommended for patients with resistant hypertension even when circulating aldosterone levels are not high.
Yujiro Maeoka   +8 more
semanticscholar   +1 more source

Effects of Nifedipine on Renal and Cardiovascular Responses to Neuropeptide Y in Anesthetized Rats

open access: yesMolecules, 2021
Neuropeptide Y (NPY) acts via multiple receptor subtypes termed Y1, Y2 and Y5. While Y1 receptor-mediated effects, e.g., in the vasculature, are often sensitive to inhibitors of L-type Ca2+ channels such as nifedipine, little is known about the role of ...
Angela Bischoff   +2 more
doaj   +1 more source

Thiazide-Sensitive NCC (Sodium-Chloride Cotransporter) in Human Metabolic Syndrome: Sodium Sensitivity and Potassium-Induced Natriuresis.

open access: yesHYPERTENSION, 2021
The thiazide-sensitive sodium-chloride cotransporter (NCC;SLC12A3) is central to sodium and blood pressure regulation. Metabolic syndrome induces NCC upregulation generating sodium-sensitive hypertension in experimental animal models.
R. Preston   +6 more
semanticscholar   +1 more source

Effects of phenylbutazone, firocoxib, and dipyrone on the diuretic response to furosemide in horses

open access: yesJournal of Veterinary Internal Medicine, 2023
Background Treatment with phenylbutazone (nonselective COX inhibitor) decreases the diuretic and natriuretic effects of furosemide by nearly 30% but the effects of COX‐2 specific inhibitors (firocoxib) and atypical NSAIDs (dipyrone) are unknown ...
Julianne M. White   +2 more
doaj   +1 more source

Novel Antihypertension Mechanism of β-Glucan by Corin and ANP-Mediated Natriuresis in Mice

open access: yesMycobiology, 2020
Many of the β-glucans are known to have antihypertensive activities, but, except for angiotensin-converting enzyme II inhibition, the underlying mechanisms remain unclear. Corin is an atrial natriuretic peptide (ANP)-converting enzyme.
Sun Jung Lee, Dong Hee Lee, Ha Won Kim
doaj   +1 more source

Renin-Angiotensin-Aldosterone System Activation and Diuretic Response in Ambulatory Patients With Heart FailurePlain-Language Summary

open access: yesKidney Medicine, 2022
Rationale & Objective: Heart failure treatment relies on loop diuretics to induce natriuresis and decongestion, but the therapy is often limited by diuretic resistance.
Jonathan G. Amatruda   +6 more
doaj   +1 more source

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