Results 11 to 20 of about 340,830 (293)

Tobacco experimental model to induce urinary bladder neoplasms [PDF]

open access: yesRevista do Colégio Brasileiro de Cirurgiões, 2014
OBJECTIVE: to develop an experimental model of exposure to tobacco burning (cigarette) products to assess the effects of its chronic use in relation to cancers of the bladder.
José Alexandre Colli Neto   +5 more
doaj   +5 more sources

Fisheye piezo polymer detector for scanning optoacoustic angiography of experimental neoplasms

open access: yesPhotoacoustics, 2023
A number of optoacoustic (or photoacoustic) microscopy and mesoscopy techniques have successfully been employed for non-invasive tumor angiography. However, accurate rendering of tortuous and multidirectional neoplastic vessels is commonly hindered by ...
Alexey Kurnikov   +6 more
doaj   +4 more sources

Experimental model of micronodular thymic neoplasm with lymphoid stroma

open access: yesThoracic Cancer, 2022
AbstractBackgroundMicronodular thymic neoplasm with lymphoid stroma (MNT), a subtype of thymic tumor, is histopathologically characterized by micronodular thymic epithelial cell nests with lymphoid stroma. Despite the distinct histopathology of MNT, its pathogenesis remains unclear.MethodsIn this study, we aimed to examine a thymic tumor harboring ...
Riko Niwa   +4 more
openaire   +3 more sources

Effect of colostomy on experimentally produced neoplasms of the colon of the rat [PDF]

open access: yesCancer, 1967
As a means of determining the effect of the fecal stream in the pathogenesis of experimentally produced colonic tumors in Wistar strain rats, simple colostomies were performed in the left colon of 57 female rats at approximately 4 cm above the anus. Fifty-four rats received daily subcutaneous injections, 5 days per week, of the carcinogen 3:2'dimethyl ...
Harlan J. Spjut, Antonio Navarrete
openaire   +3 more sources

Experimental Modeling of Myeloproliferative Neoplasms

open access: yesGenes, 2019
Myeloproliferative neoplasms (MPN) are genetically very complex and heterogeneous diseases in which the acquisition of a somatic driver mutation triggers three main myeloid cytokine receptors, and phenotypically expresses as polycythemia vera (PV), essential thrombocytosis (ET), and primary myelofibrosis (PMF).
Olga Babosova   +3 more
openaire   +2 more sources

Abnormal Expression of BTLA and CTLA-4 Immune Checkpoint Molecules in Chronic Lymphocytic Leukemia Patients

open access: yesJournal of Immunology Research, 2020
Chronic lymphocytic leukemia (CLL) is characterized by the peripheral accumulation of neoplastic B cells and is frequently complicated by the systemic immunosuppression associated with an impairment in B and T lymphocyte activation.
L. Karabon   +10 more
doaj   +1 more source

Mathematical methods and models for radiation carcinogenesis studies [PDF]

open access: yes, 1986
Research on radiation carcinogenesis requires a twofold approach. Studies of primary molecular lesions and subsequent cytogenetic changes are essential, but they cannot at present provide numerical estimates of the risk of small doses of ionizing ...
A.M. Kellerer   +25 more
core   +1 more source

MDHGI: Matrix Decomposition and Heterogeneous Graph Inference for miRNA-disease association prediction. [PDF]

open access: yesPLoS Computational Biology, 2018
Recently, a growing number of biological research and scientific experiments have demonstrated that microRNA (miRNA) affects the development of human complex diseases.
Xing Chen, Jun Yin, Jia Qu, Li Huang
doaj   +1 more source

Chromogranin A: From Laboratory to Clinical Aspects of Patients with Neuroendocrine Tumors [PDF]

open access: yes, 2018
Background. Neuroendocrine tumors (NETs) are characterized by having behavior and prognosis that depend upon tumor histology, primary site, staging, and proliferative index.
Baldelli, Roberto   +8 more
core   +1 more source

Driver mutations (JAK2V617F, MPLW515L/K or CALR), pentraxin-3 and C-reactive protein in essential thrombocythemia and polycythemia vera

open access: yesJournal of Hematology & Oncology, 2017
Background The driver mutations JAK2V617F, MPLW515L/K and CALR influence disease phenotype of myeloproliferative neoplasms (MPNs) and might sustain a condition of chronic inflammation.
Federico Lussana   +9 more
doaj   +1 more source

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