Results 41 to 50 of about 643 (155)

The Genetic Polymorphisms of HLA Are Strongly Correlated with the Disease Severity after Hantaan Virus Infection in the Chinese Han Population

open access: yesJournal of Immunology Research, Volume 2012, Issue 1, 2012., 2012
The polymorphism of human leukocyte antigen (HLA), which is a genetic factor that influences the progression of hemorrhagic fever with renal syndrome (HFRS) after Hantaan virus (HTNV) infection, was incompletely understood. In this case‐control study, 76 HFRS patients and 370 healthy controls of the Chinese Han population were typed for the HLA‐A, ‐B ...
Ying Ma   +13 more
wiley   +1 more source

Development of Type 1 Diabetes in Wild Bank Voles Associated With Islet Autoantibodies and the Novel Ljungan Virus

open access: yesJournal of Diabetes Research, Volume 4, Issue 1, Page 35-44, 2003., 2003
Wild bank voles (Clethrionomys glareolus) may develop diabetes in laboratory captivity. The aim of this study was to test whether bank voles develop type 1 diabetes in association with Ljungan virus. Two groups of bank voles were analyzed for diabetes, pancreas histology, autoantibodies to glutamic acid decarboxylase (GAD65), IA‐2, and insulin by ...
Bo Niklasson   +15 more
wiley   +1 more source

Evaluation of Real-Time RT-PCR for Diagnostic Use in Detection of Puumala Virus

open access: yesViruses, 2019
Puumala virus (PUUV) is the most common cause of hantavirus infection in Europe, with thousands of cases occurring particularly in Northern, Central and Eastern Europe and Russia.
Silja Niskanen   +3 more
doaj   +1 more source

Puumala Virus Variants Circulating in Forests of Ardennes, France: Ten Years of Genetic Evolution

open access: yesPathogens, 2021
In Europe, Puumala virus (PUUV) transmitted by the bank vole (Myodes glareolus) is the causative agent of nephropathia epidemica (NE), a mild form of haemorrhagic fever with renal syndrome.
Guillaume Castel   +12 more
doaj   +1 more source

In silico design and evaluation of a multiepitope vaccine targeting the nucleoprotein of Puumala orthohantavirus

open access: yesProteins: Structure, Function, and Bioinformatics, Volume 92, Issue 10, Page 1161-1176, October 2024.
Abstract The Puumala orthohantavirus is present in the body of the bank vole (Myodes glareolus). Humans infected with this virus may develop hemorrhagic fever accompanying renal syndrome. In addition, the infection may further lead to the failure of an immune system completely.
Kunal Bhattacharya   +4 more
wiley   +1 more source

Biomarkers in Infectious Diseases

open access: yes, 2018
Disease Markers, Volume 2018, Issue 1, 2018.
Hyundoo Hwang   +2 more
wiley   +1 more source

Demographic Factors Associated with Hantavirus Infection in Bank Voles (Clethrionomys glareolus)

open access: yesEmerging Infectious Diseases, 2002
The bank vole (Clethrionomys glareolus) is the natural reservoir of Puumala virus (PUUV), a species in the genus Hantavirus. PUUV is the etiologic agent of nephropathia epidemica, a mild form of hemorrhagic fever with renal syndrome.
Gert E. Olsson   +6 more
doaj   +1 more source

Isolation and Genetic Characterization of Puumala Orthohantavirus Strains from France

open access: yesPathogens, 2021
Puumala orthohantavirus (PUUV) causes a mild form of haemorrhagic fever with renal syndrome (HFRS) called nephropathia epidemica (NE), regularly diagnosed in Europe.
Johann Vulin   +10 more
doaj   +1 more source

New Biomarkers of Innate and Adaptive Immunity in Infectious Diseases

open access: yes, 2017
Journal of Immunology Research, Volume 2017, Issue 1, 2017.
Sergey Morzunov   +2 more
wiley   +1 more source

Risk Factors for Human Infection with Puumala Virus, Southwestern Germany

open access: yesEmerging Infectious Diseases, 2009
Puumala virus, which causes nephropathia epidemica (NE), is the most prevalent hantavirus in Germany; bank voles serve as the main reservoir. During 2001–2007, most NE cases reported from Germany occurred in the southwestern state of Baden-Württemberg ...
Anne Caroline Schwarz   +4 more
doaj   +1 more source

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