Results 181 to 190 of about 97,051 (285)
The K⁺/Cl− cotransporter (KCC2) limits induced ictal‐like events and their spread in the supragranular layer of human neocortical slices from the seizure‐propagation zone of temporal lobe epilepsy patients. Cl⁻ extrusion is present in supragranular pyramidal neurons but limited under high somatic Cl⁻ load.
Alice Falck +16 more
wiley +1 more source
A 5‐month‐old infant with a pineal tumor
Brain Pathology, EarlyView.
Fabien Forest +7 more
wiley +1 more source
Olig2‐specific loss‐of‐function Slc35a2 results in hypomyelination and spontaneous seizures
Abstract Objective Malformations of cortical development represent major causes of drug‐resistant epilepsy, with mild malformation of cortical development with oligodendroglial hyperplasia and epilepsy recognized as a distinct pathological entity. Pathogenic X‐linked SLC35A2, encoding the uridine diphosphate–galactose transporter, has been implicated ...
Tiffany M. Bartel +6 more
wiley +1 more source
APOE4 to APOE2 ‘Switching’ in the CNS vs Periphery: Differential Effects on AD Neuropathology [PDF]
Lance A. Johnson
openalex +1 more source
A 17‐year‐old male with chronic epilepsy status post remote functional hemispherectomy
Brain Pathology, EarlyView.
Claire Voyles +3 more
wiley +1 more source
The epileptologist's perspective of focal cortical dysplasia type 3: From concept to management
Abstract The recent International League Against Epilepsy (ILAE) official and updated classification of focal cortical dysplasia (FCD) includes a third type—FCD type 3—characterized by architectural abnormalities (cortical dyslamination) associated with another “principal” lesion: hippocampal sclerosis (HS), developmental tumors, vascular malformations,
André Palmini +10 more
wiley +1 more source
Subtypes of multiple-etiology dementias and the heterogeneous impact of APOE variants. [PDF]
Pirraglia E +4 more
europepmc +1 more source

