Results 241 to 250 of about 219,450 (292)
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Physiology and pathophysiology of purinergic neurotransmission.
Physiological Reviews, 2007This review is focused on purinergic neurotransmission, i.e., ATP released from nerves as a transmitter or cotransmitter to act as an extracellular signaling molecule on both pre- and postjunctional membranes at neuroeffector junctions and synapses, as ...
G. Burnstock
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Neurotransmitters and Neurotransmission
2021Normal functioning of the human brain requires parallel and sequential innumerous neurotransmission processes that are fine-tuned by countless organic chemicals called neurotransmitters. Different neurotransmitters are expressed at a higher level in distinct areas of the brain signifying their physiological role.
Mageshwaran Lakshmanan, Elavarasi Pichai
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ACS Nano, 2020
Neurotransmission is the basis of brain functions, and controllable neurotransmission tuning constitutes an attractive approach for interventions in a wide range of neurologic disorders and for synapse-based therapeutic treatments.
Yiyuan Kang+9 more
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Neurotransmission is the basis of brain functions, and controllable neurotransmission tuning constitutes an attractive approach for interventions in a wide range of neurologic disorders and for synapse-based therapeutic treatments.
Yiyuan Kang+9 more
semanticscholar +1 more source
Annual Review of Nutrition, 2011
A subset of glutamatergic synapses in the central nervous system contains zinc; it is sequestered into the lumen of synaptic vesicles, where it colocalizes with glutamate. Extracellularly applied zinc is known to interact with various postsynaptic receptors and channels; however, the role of endogenous vesicular zinc is still an enigma.
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A subset of glutamatergic synapses in the central nervous system contains zinc; it is sequestered into the lumen of synaptic vesicles, where it colocalizes with glutamate. Extracellularly applied zinc is known to interact with various postsynaptic receptors and channels; however, the role of endogenous vesicular zinc is still an enigma.
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Neurotransmission In The Auditory System
Otolaryngologic Clinics of North America, 1992Neurotransmitters and neuromodulators thought to be active on neurons in the cochlea, CN, and SOC have been reviewed. The variety of neurotransmitters and neuromodulators present and likely colocalized in these neurons are the chemical substrates that link morphologically and physiologically diverse neurons to process sound information.
Robert J. Wenthold+2 more
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Epilepsia, 1995
Summary: Some evidence indicates that in some types of focal epilepsy the enhanced excitability is due in part to impaired γ‐aminobutyric acid (GABA)ergic inhibitory feedback. One form that this can take is impaired excitatory input to GABAergic interneurons.
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Summary: Some evidence indicates that in some types of focal epilepsy the enhanced excitability is due in part to impaired γ‐aminobutyric acid (GABA)ergic inhibitory feedback. One form that this can take is impaired excitatory input to GABAergic interneurons.
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Neurotransmission: sites at which drugs modify neurotransmission
1983The idea that nerves may communicate with other cells by releasing small quantities of chemicals at their junctions may have arisen from observations of the effects of poisons on animals. It was found that some poisons could mimic the effects of stimulating certain nerves, and it must have occurred to somebody that nerves release chemicals in response ...
Zygmunt L. Kruk, Christopher J. Pycock
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