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Memantine, an NMDA Receptor Antagonist, Attenuates Doxorubicin-Induced Cardiac Oxidative Stress and Inflammation in Mouse 4T1 Breast Cancer Model. [PDF]
Ghafouri-Asbagh A +6 more
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Spike Timing-Dependent Plasticity at Layer 2/3 Horizontal Connections Between Neighboring Columns During Synapse Formation Before the Critical Period in the Developing Barrel Cortex. [PDF]
Itami C, Kimura F.
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Lifelong Impact of Prenatal Stress: Exacerbated Memory Impairments and Gene Expression Changes Under Adult Chronic Stress. [PDF]
Dogani M, Askari N, Vaez-Mahdavi MR.
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Varón joven con encefalitis autoinmune por anticuerpos anti receptor NMDA
Milthon Blanco-Mamani +2 more
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Desenvolvimento de novos antagonistas do recetor NMDA
Nuno Alexandre Lousa Pereira
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Down-regulation of NMDA receptor activity by NMDA
Neuroscience Letters, 1993Rat cerebellar granule cells were cultured in a medium containing 25 mM KCl. The presence of NMDA during culture caused strong down-regulation of 45Ca uptake through the NMDA receptor channel. The process affected neither the viability nor the protein content of the cells. The developmental program of NMDA receptor activity was resumed after removal of
Y, Oster, M, Schramm
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The Neuroscientist, 2007
The amino acid L-Glutamate acts as the most ubiquitous mediator of excitatory synaptic transmission in the central nervous system. Glutamatergic transmission is central for diverse brain functions, being particularly important for learning, memory, and cognition. In brain pathology, excessive release of glutamate triggers excitotoxic neural cell death
Verkhratsky, Alexei, Kirchhoff, Frank
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The amino acid L-Glutamate acts as the most ubiquitous mediator of excitatory synaptic transmission in the central nervous system. Glutamatergic transmission is central for diverse brain functions, being particularly important for learning, memory, and cognition. In brain pathology, excessive release of glutamate triggers excitotoxic neural cell death
Verkhratsky, Alexei, Kirchhoff, Frank
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Journal of Neurochemistry, 1994
Abstract:Transient forebrain or global ischemia in rats induces selective and delayed damage of hippocampal CA1 neurons. In a previous sludy, we have shown that expression of GIuR2, the kainate/a‐amino‐3‐hydroxy‐5‐ methyl‐4‐isoxazolepropionic acid (AMPA) receptor subunit that governs Ca′ permeability, is preferentially reduced in CA1 at a time point ...
PELLEGRINI-GIAMPIETRO, DOMENICO EDOARDO +2 more
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Abstract:Transient forebrain or global ischemia in rats induces selective and delayed damage of hippocampal CA1 neurons. In a previous sludy, we have shown that expression of GIuR2, the kainate/a‐amino‐3‐hydroxy‐5‐ methyl‐4‐isoxazolepropionic acid (AMPA) receptor subunit that governs Ca′ permeability, is preferentially reduced in CA1 at a time point ...
PELLEGRINI-GIAMPIETRO, DOMENICO EDOARDO +2 more
openaire +3 more sources

