Results 11 to 20 of about 4,862 (192)

Inadvertent Extravasations of Norepinephrine

open access: yesClinical Case Reports, 2022
A 63-year-old male patient of diabetic ketoacidosis and septic shock was started on norepinephrine infusion following which he developed bulla and subcutaneous tissue ischemia in the event of inadvertent extravasations of norepinephrine. The patient improved after management with mechanical debridement of necrosed tissue and regular dressing of the ...
openaire   +3 more sources

A cautionary note for researchers treating mice with the neurotransmitter norepinephrine

open access: yesBiochemistry and Biophysics Reports, 2018
The sympathetic nervous system plays a crucial role in metabolic function and glucose homeostasis. Norepinephrine is the main neurotransmitter released from sympathetic neurons. The major goal of our studies was to examine the impact of norepinephrine on
Vance B. Matthews   +2 more
doaj   +1 more source

The Impact of Norepinephrine on Mono-Species and Dual-Species Staphylococcal Biofilms

open access: yesMicroorganisms, 2021
The effect of norepinephrine (“NE”) on Gram-negative bacteria is well characterized; however, little is known about the impact of NE on cutaneous Gram-positive skin residents, especially staphylococci.
Sergey Vladislavovich Mart’yanov   +3 more
doaj   +1 more source

Norepinephrine-evoked pain in fibromyalgia. A randomized pilot study [ISRCTN70707830]

open access: yesBMC Musculoskeletal Disorders, 2002
Background Fibromyalgia syndrome displays sympathetically maintained pain features such as frequent post-traumatic onset and stimuli-independent pain accompanied by allodynia and paresthesias.
Casanova Jose-Miguel   +5 more
doaj   +1 more source

Impaired astrocytic Ca2+ signaling in awake-behaving Alzheimer’s disease transgenic mice

open access: yeseLife, 2022
Increased astrocytic Ca2+ signaling has been shown in Alzheimer’s disease mouse models, but to date no reports have characterized behaviorally induced astrocytic Ca2+ signaling in such mice.
Knut Sindre Åbjørsbråten   +14 more
doaj   +1 more source

Norepinephrine as a Potential Aggravator of Symptomatic Cerebral Vasospasm: Two Cases and Argument for Milrinone Therapy

open access: yesCase Reports in Critical Care, 2014
Background. During hypertensive therapy for post-subarachnoid hemorrhage (SAH) symptomatic vasospasm, norepinephrine is commonly used to reach target blood pressures. Concerns over aggravation of vasospasm with norepinephrine exist. Objective.
F. A. Zeiler   +4 more
doaj   +1 more source

Molecular effects of intermittent stress on primary feline uroepithelial cell culture as an in vitro model of feline idiopathic cystitis

open access: yesFrontiers in Veterinary Science, 2023
IntroductionThe most common cause of feline lower urinary tract disease (FLUDT) is feline idiopathic cystitis (FIC), which is a complex multifactorial disease with symptoms including stranguria, dysuria, hematuria, and pain during urination.
Patrícia Hatala   +8 more
doaj   +1 more source

Vasopressin versus norepinephrine in septic shock: a propensity score matched efficiency retrospective cohort study in the VASST coordinating center hospital

open access: yesJournal of Intensive Care, 2018
Purpose It is not clear whether vasopressin versus norepinephrine changed mortality in clinical practice in the Vasopressin and Septic Shock Trial (VASST) coordinating center hospital after VASST was published.
James A. Russell   +2 more
doaj   +1 more source

Sympathetic hyperactivity in patients with hypertension: pathogenesis and treatment. Part II

open access: yesКардиоваскулярная терапия и профилактика, 2021
The second part of the review considers different classes of drugs affecting blood pressure in increased activity of the sympathetic nervous system. Additional possibilities are discussed on how to reduce the negative effect of sympathetic hyperactivity ...
A. S. Safaryan, D. V. Nebieridze
doaj   +1 more source

TYRAMINE REVEALS FAILING α2-ADRENOCEPTOR CONTROL OF CATECHOLAMINE RELEASE AND TOTAL PERIPEHRAL VASCULAR RESISTANCE IN HYPERTENSIVE RATS

open access: yesFrontiers in Neurology, 2013
α2-adrenoceptor-activation lowers central sympathetic output, peripheral, vesicular norepinephrine release, epinephrine secretion and modulates vascular tension.
Torill eBerg, Jørgen eJensen
doaj   +1 more source

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