A secretory hexokinase plays an active role in the proliferation of Nosema bombycis [PDF]
PeerJ, 2018 The microsporidian Nosema bombycis is an obligate intracellular parasite of Bombyx mori, that lost its intact tricarboxylic acid cycle and mitochondria during evolution but retained its intact glycolysis pathway. N. bombycis hexokinase (NbHK) is not only
Yukang Huang +11 more
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Stable transformation of fluorescent proteins into Nosema bombycis by electroporation [PDF]
Parasites & Vectors, 2022 Background Microsporidia are a group of intracellular parasitic eukaryotes, serious pathogens that cause widespread infection in humans, vertebrates, and invertebrates. Because microsporidia have a thick spore wall structure, the in vitro transformation,
Zhanqi Dong +8 more
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Quantitative proteomic analysis of germination of Nosema bombycis spores under extremely alkaline conditions [PDF]
Frontiers in Microbiology, 2016 The microsporidian Nosema bombycis is an obligate intracellular pathogen of the silkworm Bombyx mori, causing the epidemic disease Pebrine and extensive economic losses in sericulture. Although N. bombycis forms spores with rigid spore walls that protect
Yongqi Shao
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Maturation of subtilisin-like protease NbSLP1 from microsporidia Nosema bombycis [PDF]
Frontiers in Cellular and Infection Microbiology, 2022 Microsporidia are obligate intracellular parasites and possess a unique way of invading hosts, namely germination. Microsporidia are able to infect almost all animal cells by germination. During the process, the polar tube extrudes from the spores within,
Rong Wang +11 more
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Functional characterization of an aquaporin from a microsporidium, Nosema bombycis. [PDF]
PLoS ONE, 2017 Microsporidia are a diverse group of eukaryotic organisms, capable of causing parasitic infections in both vertebrates and invertebrates. During the germination process, there is an increase in the osmotic pressure of microsporidian spores.
Gong Chen +8 more
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A monoclonal antibody that tracks endospore formation in the microsporidium Nosema bombycis. [PDF]
PLoS ONE, 2015 Nosema bombycis, the first identified microsporidium, is a destructive pathogen of the silkworm Bombyx mori and causes severe worldwide economic losses in sericulture.
Yanhong Li +5 more
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A monoclonal antibody targeting spore wall protein 1 inhibits the proliferation of Nosema bombycis in Bombyx mori [PDF]
Microbiology Spectrum, 2023 Microsporidia are obligate intracellular single-cell eukaryotic parasites that can infect almost all kinds of animals and cause microsporidiosis. However, there are a few achievements of microsporidiosis treatments.
Bin Yu +8 more
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Detection and Characterization of Nosema bombycis Using TEM and SEM Techniques. [PDF]
Arch Razi Inst, 2022 Pebrine disease is the most important and dangerous disease of silkworm caused by Nosema bombycis as an obligate intracellular parasitic fungus. It has caused tremendous economic losses in the silk industry in recent years. Given the fact that light microscopy method (with low accuracy) is the only method for diagnosing pebrine disease in the country ...
Moharrami M, Bagheri M, Nematollahian S.
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The gut commensal bacterium Enterococcus faecalis LX10 contributes to defending against Nosema bombycis infection in Bombyx mori. [PDF]
Pest Manag Sci, 2022 According to the microsporidia–Bombyx mori model, we revealed that Enterococcus with anti‐Nosema bombycis activity might play an important role in protecting silkworms from microsporidia. The present study provides a clear profile of the complex interplay among microsporidia, silkworms, and gut microbiome.
Zhang X +7 more
europepmc +2 more sources
Nosema bombycis microRNA-like RNA 8 (Nb-milR8) Increases Fungal Pathogenicity by Modulating BmPEX16 Gene Expression in Its Host, Bombyx mori [PDF]
Microbiology Spectrum, 2021 The fungus Nosema bombycis causes significant economic losses via parasitism of an economically important insect. MicroRNAs (miRNAs) play important roles in regulating host and parasite gene expression via mRNA degradation or by inhibiting protein ...
Zhanqi Dong +10 more
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