Results 81 to 90 of about 67,803 (296)
Ocular surface disease in patients with glaucoma or ocular hypertension treated with either BAK-preserved latanoprost or BAK-free travoprost [PDF]
Gregory Katz1, Clark L Springs2, E Randy Craven3, Michela Montecchi-Palmer41Huron Ophthalmology, Ypsilanti, MI, USA; 2Indiana University Eye Care, Indianapolis, IN, USA; 3Specialty Eye Care, Denver, CO, USA; 4Alcon Research Ltd., Fort Worth, TX ...
Clark L Springs +3 more
core
Summary of existing surgically-induced rodent ocular hypertension models. [PDF]
Summary of existing surgically-induced rodent ocular hypertension models.
Nalini Rangaswamy (5460071) +6 more
core +1 more source
The role of central corneal thickness in the diagnosis of glaucoma
Purpose: To determine the effect of central corneal thickness (CCT) on applanation tonometry and any resultant misclassification of normals as ocular hypertension.
Thomas Ravi +2 more
doaj
Therapeutic problems in ocular hypertension
Sood N, Grover A, Agarwal H
doaj +2 more sources
Canagliflozin treatment reshapes the gut microbiota in DKD and elevates levels of melibiose, a metabolite derived from Roseburia intestinalis. Melibiose directly binds to and enhances the enzymatic activity of glyoxalase 1, leading to decreased methylglyoxal accumulation.
Wei Zhang +32 more
wiley +1 more source
ORIGINAL STUDY Lifetime Visual Disability in Open-angle Glaucoma [PDF]
Purpose: To study how many of the patients with treated glaucoma or ocular hypertension go blind during their lifetime and which factors are associated with blindness.
Ocular Hypertension +3 more
core
Nisa Silva,1 André Ferreira,1,2 Pedro Manuel Baptista,1 Ana Figueiredo,1 Rita Reis,1 Isabel Sampaio,1 João Beirão,1,3 Riccardo Vinciguerra,4,5 Pedro Menéres,1,3 Maria João Menéres1,3 1Ophthalmology Department, Centro Hospitalar Universitário do Porto ...
Silva N +9 more
doaj
Endogenous hypercortisolism inducing reversible ocular hypertension
Purpose: To describe the clinical findings of two patients with reversible ocular hypertension secondary to endogenous hypercortisolism. Design: Retrospective, observational case series.
Shane Griffin +5 more
doaj +1 more source
This diagram illustrates that night shift work disrupts circadian clock genes (like CLOCK, BMAL1) in both humans and mice. This disruption leads to mitochondrial dysfunction (imbalanced fusion/fission proteins) and increased oxidative stress, which is identified as the primary mechanism ultimately causing elevated blood pressure.
Zhaoqiang Jiang +16 more
wiley +1 more source
The Accuracy and Clinical Application of Predictive Models for Primary Open-Angle Glaucoma in Ocular Hypertensive Individuals [PDF]
[. . . ] +6 more
core +3 more sources

