Results 131 to 140 of about 577,954 (290)
Transformation by the fos or jun oncogene does not increase AP-1 DNA-binding activity [PDF]
Kelvin Hawker +4 more
openalex +1 more source
Epstein‐Barr Virus Expressed Long Non‐Coding RNA (lncBARTs) Regulate EBV Latent Genome Replication
EBV produces abundant level of lncBARTs, which are essential for maintaining viral genome replication in EBV‐associated cancers. LncBARTs interact with a complex comprising BRD4, CTCF and viral protein EBNA1 at EBV oriP region. This interaction tethers oriP to host chromosomes, facilitating EBV episome replication.
Jiayan Liu +12 more
wiley +1 more source
RNA Methylation in Cancer Metabolism: from Mechanisms to Therapeutic Opportunities. [PDF]
Wu Z +5 more
europepmc +1 more source
Microbial Secondary Metabolites Inhibiting Oncogene Functions.
Kazuo Umezawa
openalex +2 more sources
METTL1 and its mediated m7G tRNA modification are significantly up‐regulated after partial hepatectomy (PHx). Overexpression of METTL1 improves the efficiency of liver regeneration after PHx, increases the proliferation of hepatocytes, and accelerates the recovery of liver function.
Manling Huang +18 more
wiley +1 more source
MicroRNAs as the critical regulators of ubiquitin-proteasome system through F-box protein targeting during tumor progression. [PDF]
Akhlaghipour I, Moghbeli M.
europepmc +1 more source
Hunting the elusive oncogene: a stroke of good luck [PDF]
Robert A. Weinberg
openalex +1 more source
Oxidative phosphorylation (OXPHOS) activity serves as a critical metabolic determinant of radiotherapy outcomes in esophageal squamous cell carcinoma (ESCC). The CEBPB/AREG/EREG axis drives radioresistance in ESCC by reprogramming OXPHOS. AREG and EREG function as predictive biomarkers for nCRT response and prognosis of ESCC patients.
Zhang Lin +15 more
wiley +1 more source
[Retracted] 8‑bromo‑7‑methoxychrysin induces apoptosis by regulating Akt/FOXO3a pathway in cisplatin‑sensitive and resistant ovarian cancer cells [PDF]
Ding Q +6 more
europepmc +1 more source

