Results 111 to 120 of about 305,335 (274)
Librarians amongst many others point towards Open Access Publishing as being the loophole in the current library and journal crisis. However, Open Access Publishing could entail higher costs for the university as a whole even though spendings for ...
Bauer, Bruno
doaj
Open access: Māori perspectives on publishing
Introduction. This short paper presents the results of a project that looked at Māori researchers’ attitudes to open access publishing. It presents an overview of their engagement with green and gold publishing options, including barriers and challenges
Spencer Lilley
doaj +1 more source
Following high dose rate brachytherapy (HDR‐BT) for hepatocellular carcinoma (HCC), patients were classified as responders and nonresponders. Post‐therapy serum induced increased BrdU incorporation and Cyclin E expression of Huh7 and HepG2 cells in nonresponders, but decreased levels in responders.
Lukas Salvermoser +14 more
wiley +1 more source
OPERAS: bringing the long tail of Social Sciences and Humanities into Open Science
The paper will present OPERAS, a comprehensive infrastructure aimed at providing a pan-European infrastructure to rethink and reshape publishing, discovery and dissemination addressing the specificity and the critical issues of Social Sciences and ...
Elena Giglia
doaj +2 more sources
Principles of Best Practice and Transparency in Scholarly Publishing ver. 4: a Korean translation [PDF]
The Committee on Publication Ethics, DOAJ, the Open Access Scholarly Publishing Association, the World Association of Medical Editors
doaj +1 more source
Moving to the forefront of open access publishing in dermatology. [PDF]
Langan EA.
europepmc +1 more source
Inhibition of CDK9 enhances AML cell death induced by combined venetoclax and azacitidine
The CDK9 inhibitor AZD4573 downregulates c‐MYC and MCL‐1 to induce death of cytarabine (AraC)‐resistant AML cells. This enhances VEN + AZA‐induced cell death significantly more than any combination of two of the three drugs in AraC‐resistant AML cells.
Shuangshuang Wu +18 more
wiley +1 more source
A synthetic benzoxazine dimer derivative targets c‐Myc to inhibit colorectal cancer progression
Benzoxazine dimer derivatives bind to the bHLH‐LZ region of c‐Myc, disrupting c‐Myc/MAX complexes, which are evaluated from SAR analysis. This increases ubiquitination and reduces cellular c‐Myc. Impairing DNA repair mechanisms is shown through proteomic analysis.
Nicharat Sriratanasak +8 more
wiley +1 more source
Behavioral Reluctance in Adopting Open Access Publishing: Insights From a Goal-Directed Perspective. [PDF]
Köster M +5 more
europepmc +1 more source

