Results 211 to 220 of about 56,247 (306)

Luteinizing hormone receptor knockout mouse: What has it taught us?

open access: yesAndrology, Volume 14, Issue 4, Page 1017-1028, May 2026.
Abstract Luteinizing hormone (LH), along with its agonist choriongonadotropin (hCG) in humans, is the key hormone responsible for the tropic regulation of the gonadal function. LH and hCG act through their cognate receptor, the luteinizing hormone/choriongonadotropin receptor (LHCGR; more appropriately LHR in rodents lacking CG), located in the testis ...
Ilpo T. Huhtaniemi
wiley   +1 more source

A Population‐Based Study of Posterior Reversible Encephalopathy Syndrome and Reversible Cerebral Vasoconstriction Syndrome During Pregnancy and Puerperium

open access: yesEuropean Journal of Neurology, Volume 33, Issue 5, May 2026.
In this nationwide, population‐based study, 27 cases of posterior reversible encephalopathy syndrome (PRES) and reversible cerebral vasoconstriction syndrome (RCVS) during pregnancy or the puerperium were identified from Finnish national healthcare registers (1987–2016), with stroke occurring in 40.7% of cases.
Vest Teresa   +9 more
wiley   +1 more source

Characteristics of a <i>Dinophysis</i> cf <i>acuminata</i> Population from a Tidewater Glacier Lagoon in a Temperate Latitude: Applications to <i>Dinophysis</i> Studies. [PDF]

open access: yesMar Drugs
Díaz PA   +10 more
europepmc   +1 more source

Animal Models of Hunner‐Type Interstitial Cystitis

open access: yesInternational Journal of Urology, Volume 33, Issue 5, May 2026.
ABSTRACT Background Animal models are crucial for mechanistic studies and therapeutic development of human diseases. At present, the etiology of interstitial cystitis/bladder pain syndrome (IC/BPS), a chronic disease of the urinary bladder, remains undefined.
Yoshiyuki Akiyama, Yi Luo
wiley   +1 more source

Mixed Signals: T Cells as Architects of IgE Immunity

open access: yesImmunological Reviews, Volume 339, Issue 1, May 2026.
ABSTRACT Food allergen‐specific IgE can cause significant pathology, yet the pathways that generate pathogenic, high‐affinity IgE remain incompletely understood. Increasing evidence suggests that IgE responses arise from the integration of multiple, and sometimes opposing, T cell–derived cues.
Abigail L. Tierney   +3 more
wiley   +1 more source

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