Results 301 to 310 of about 557,753 (336)

Pentraxin3 exacerbates acute pancreatitis injury by inhibiting oxidative phosphorylation pathway. [PDF]

open access: yesSci Rep
Wang W   +10 more
europepmc   +1 more source

Clinical and Genetic Insights Into Combined Oxidative Phosphorylation Defect Type 38. [PDF]

open access: yesClin Case Rep
Yadav BK   +6 more
europepmc   +1 more source
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Oxidative phosphorylation revisited

Biotechnology and Bioengineering, 2015
ABSTRACTThe fundamentals of oxidative phosphorylation and photophosphorylation are revisited. New experimental data on the involvement of succinate and malate anions respectively in oxidative phosphorylation and photophosphorylation are presented. These new data offer a novel molecular mechanistic explanation for the energy coupling and ATP synthesis ...
Sunil, Nath, John, Villadsen
openaire   +2 more sources

Mitochondrial Oxidative Phosphorylation

Angewandte Chemie International Edition in English, 1967
AbstractMitochondria can form ATP from ADP and inorganic phosphate, the required energy being supplied by respiration. This coupled process, which in sufficiently aerated normal animal cells furnishes the bulk of the cellular ATP, is termed oxidative phosphorylation.
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Non-phosphorylated intermediates in oxidative phosphorylation

Biochimica et Biophysica Acta, 1963
Abstract 1. 1. This paper is concerned with the study of the formation of non-phosphorylated intermediates in digitonin particles prepared from rat-liver mitochondria. 2. 2. Digitonin particles were preincubated with either β-hydroxybutyrate, sucinate or ferrocytochrome c for a short time at 20°.
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Mechanism of Oxidative Phosphorylation

Nature, 1958
THE phenothiazine tranquillizer chlorpromazine has the following actions: (1) It inhibits electron transport between reduced diphosphopyridine nucleotide and cytochrome c in appropriate systems; the efficiency of the coupled phosphorylation is not affected. (2) Chlorpromazine is not effective in mitochondria treated with water or in non-phosphorylating
M J, DAWKINS, J D, JUDAH, K R, REES
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Efficiency of Oxidative Phosphorylation

Nature, 1954
THE quantitative yield of phosphate fixed during cellular oxidations deserves critical investigation, for it determines the efficiency with which utilizable energy may be derived from assimilated foodstuffs and it may influence metabolic regulatory mechanisms1. This problem has been the subject of many investigations (cf. Krebs et al.
H, LARDY, J H, COPENHAVER
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Oxidative Phosphorylation in Mitochondria

Annual Review of Biochemistry, 1969
INTRODUCTION. • • • • • • • • • • • • • • • • • • • • • • • • • • • • . • • • • • • • • • • • • • • • • • • • • • • • • • • • • • • 991 ELECTRON TRANSPORT AND COUPLING SITES. • • • • • • • • • • • • • • • • • • • • • • • • • • • • • • • • • 991 PROTEIN FACTORS THAT COUPLE RESPIRATION WITH PHOSPHORYLATION. • • • • • • • • • • 999 Phosphoryl tran rerases.
H A, Lardy, S M, Ferguson
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Oxidative phosphorylation and aging

Ageing Research Reviews, 2006
This review addresses the data that support the presence and contribution of decreased mitochondrial oxidative phosphorylation during aging to impaired cellular metabolism. Aging impairs substrate oxidation, decreases cellular energy production and increases the production of reactive intermediates that are toxic to the cell.
Edward J, Lesnefsky, Charles L, Hoppel
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