Results 211 to 220 of about 24,766 (261)

Effect Of P. Gingivalis Outer Membrane Vesicles In Preventing Periodontitis

International Dental Journal
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P. gingivalis interactions with epithelial cells

Frontiers in Bioscience, 2008
Dental plaque, a microbial biofilm that accumulates on teeth and initiates periodontal disease, is composed of hundreds of different bacterial species within an organized structure. The biofilm bacteria and their byproducts irritate the gingival epithelium and induce an "inflammatory response".
Denis F, Kinane, Johnah Cortez, Galicia, Sven-Ulrik, Gorr, Panagiota Giorgios, Stathopoulou, Manjunatha, Benakanakere   +4 more
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Theaflavins inhibit pathogenic properties of P. gingivalis and MMPs production in P. gingivalis-stimulated human gingival fibroblasts

Archives of Oral Biology, 2015
Theaflavins, the main polyphenols in black tea, possesses a wide range of beneficial pharmacological properties. Porphyromonas gingivalis (P. gingivalis) is a major aetiological agent associated with periodontitis, a chronic inflammatory disease affecting tooth-supporting tissues. The aim of the present study is to investigate the effect of theaflavins
Lingxue, Kong, Xia, Qi, Shiyan, Huang, Shanshan, Chen, Yafei, Wu, Lei, Zhao   +5 more
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P. gingivalis drives carcinoma progression

Nature Reviews Microbiology, 2020
This study found that the oral pathogen Porphyromonas gingivalis promotes the progression of oesophageal squamous cell carcinoma.
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Anti- P. gingivalis Response Correlates with Atherosclerosis

Journal of Dental Research, 2007
Significant associations between atherosclerosis and both Porphyromonas gingivalis, a major periodontopathogen, and the respiratory pathogen, Chlamydia pneumoniae, have been shown. Many individuals with evidence of atherosclerosis demonstrate seropositivity to these pathogens.
Ford, Pauline, Gemmell, Edward, Timms, Peter, Chan, A, Preston, F, Seymour, G   +5 more
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Effects of Tobacco and P. gingivalis on Gingival Fibroblasts

Journal of Dental Research, 2010
Cigarette smoke condensate (CSC) is the particulate matter of cigarette smoke. Porphyromonas gingivalis ( P. gingivalis) is an opportunistic pathogen involved in periodontitis. It was hypothesized that the combination of CSC and P. gingivalis would increase the collagen-degrading ability of human gingival fibroblasts (HGFs).
W, Zhang, F, Song, L J, Windsor
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Variable Cell Responses to P. gingivalis Lipopolysaccharide

Journal of Dental Research, 2009
Porphyromonas gingivalis is a major etiological agent of chronic periodontal diseases, the virulence of which has been attributed to different factors, including lipopolysaccharide (LPS). We investigated the differential responses induced by P. gingivalis LPS stimulation of human umbilical vein endothelial cells and human oral epithelial cells.
L, Kocgozlu, R, Elkaim, H, Tenenbaum, S, Werner   +3 more
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High Antibody Levels to P. gingivalis in Cardiovascular Disease

Journal of Dental Research, 2010
Recent evidence suggests that strain variation in the serum IgG response to Porphyromonas gingivalis occurs in periodontal disease and cardiovascular disease (CVD). This study aimed to test the hypothesis that different P. gingivalis strains would elicit different levels of IgG, depending on a patient’s cardiovascular (CV) and periodontal health.
Bohnstedt, S., Cullinan, M. P., Ford, P. J., Palmer, J. E., Leishman, S. J., Westerman, B., Marshall, R. I., West, M. J., Seymour, G. J.   +8 more
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