Results 221 to 230 of about 24,766 (261)

[Effect of A.viscosus on growth of P. gingivalis in vitro].

Shanghai kou qiang yi xue = Shanghai journal of stomatology, 2015
To investigate the effect of A. viscosus on growth of P. gingivalis in vitro.Groups as single-species(A.viscosus or P.gingivalis) and dual-species (A.viscosus and P.gingivalis) were incubated in BHI broth anaerobically at 37 degrees C, and growth curve of each group was monitored for 48 hours; Meanwhile the dual-species cultures of 2-hour interval were
Yan, Miao, Miao, Yu, Yao-lun, Han, Yu-qing, Hao   +3 more
openaire   +2 more sources

The involvement of Kav001 in inhibition of LPS/P. gingivalis‐induced

Journal of Cellular Biochemistry, 2018
AbstractTNF‐a is an important cytokine mediator of inflammation which suggests that inhibition of TNF activity may provide potential for clinical application. Recent data indicated that treatment of both human and mouse cells with Kavain significantly modulates P. gingivalis‐ and LPS‐induced TNF‐α expression.
Xiaoren Tang, Mansour Alasiri, Abdullah Bamashmous, Bushra Aljahdali, Feng Cao, Serge Dibart, Erdjan Salih   +6 more
openaire   +2 more sources

Oral infection with P. gingivalis exacerbates autoimmune encephalomyelitis

Journal of Periodontology, 2018
AbstractBackgroundOral infection of mice with P. gingivalis induces periodontal inflammation and attachment loss. The aim of the present study was to investigate whether infection of mice with P. gingivalis, exacerbates the clinical course of experimental autoimmune encephalomyelitis (EAE)—a mouse model of multiple sclerosis (MS).MethodsInduction of ...
David, Polak, Aviv, Shmueli, Talma, Brenner, Lior, Shapira   +3 more
openaire   +2 more sources

Ckip-1 Mediates P. gingivalis –Suppressed Cementoblast Mineralization

Journal of Dental Research, 2021
Porphyromonas gingivalis is responsible for the destruction of cementum in patients with periodontitis and periapical periodontitis. However, research about the effects of P. gingivalis on cementoblast mineralization and the underlying mechanism is still lacking.
X. Huang, L. Ma, X. Wang, H. Wang, Y. Peng, X. Gao, H. Huang, Y. Chen, Y. Zhang, Z. Cao   +9 more
openaire   +2 more sources

Porphyromonas gingivalis fimbriae are pro-inflammatory but do not play a prominent role in the innate immune response to P. gingivalis

Journal of Endotoxin Research, 2005
The fimA gene encodes the major fimbrial protein of Porphyromonas gingivalis. It has been shown to stimulate adhesion to salivary proteins and other bacteria. It is also thought to play a major role in invading and stimulating host cells. To determine whether the fimA gene represents one of the principal molecules of P.
Dana T, Graves, Ghada, Naguib, Huafei, Lu, Tesfahun, Desta, Salomon, Amar   +4 more
openaire   +2 more sources

Role of Porphyromonas gingivalis 40-kDa outer membrane protein in the aggregation of P. gingivalis vesicles and Actinomyces viscosus

Archives of Oral Biology, 1992
Porphyromonas gingivalis, an important pathogen in periodontitis, produces extracellular vesicles that aggregate with Actinomyces viscosus cells. A 40-kDa outer membrane protein (OMP)-coding gene from P. gingivalis was cloned and the protein was found to be localized in these vesicles.
K, Hiratsuka, Y, Abiko, M, Hayakawa, T, Ito, H, Sasahara, H, Takiguchi   +5 more
openaire   +2 more sources

The prevalence of A. actinomycetemcomitans, P. gingivalis and P. intermedia in selected subjects with periodontitis

Journal of Clinical Periodontology, 1994
Abstract. The present study was carried out in a private periodontal practice. 8 clinical criteria were chosen to select patients for microbiological examination. These criteria characterize prominent features of disease which at the start, during, or in the maintenance phase of treatment suggest difficulties in the progress of the periodontal therapy.
G A, Van der Weijden, M F, Timmerman, E, Reijerse, G N, Wolffe, A J, Van Winkelhoff, U, Van der Velden   +5 more
openaire   +2 more sources

[The impact of S.gordonii on P. gingivalis on the form of biofilm].

Shanghai kou qiang yi xue = Shanghai journal of stomatology, 2016
To investigate the impact of S.gordonii on the ultrastructure of P. gingivalis biofilm and on the amount of P. gingivalis in biofilm.P. gingivalis and/or S.gordonii grew on the culture slides to form single P. gingivalis biofilm and heterotypic biofilm of P.gingivalis-S.gordonii.
Dong-mei, Zhang, Jing-bo, Liu, Ya-ping, Pan, Jia-yu, Pan, Qiu-fang, Xu   +4 more
openaire   +1 more source

P. gingivalis Regulates the Expression of Cathepsin B and Cystatin C

Journal of Dental Research, 2008
Porphyromonas gingivalis is a major etiological agent of periodontitis that could affect the expression of Cathepsins B and C by disrupting the balance between these enzymes and their inhibitor, Cystatin C. We tested this hypothesis by infecting human oral epithelial cells with P. gingivalis or activating solely by its lipopolysaccharide.
R, Elkaim, S, Werner, L, Kocgozlu, H, Tenenbaum   +3 more
openaire   +2 more sources

Dendritic-NK Cell Interactions in P. gingivalis -specific Responses

Journal of Dental Research, 2005
Patients with localized aggressive periodontitis have type-1 cytokines in gingival crevicular fluid and high titers of IFN-γ-dependent IgG2 reactive with P. gingivalis in gingival crevicular fluid and serum. Localized aggressive periodontitis monocytes spontaneously differentiate into dendritic cells that can stimulate IFN-γ production by NK cells ...
T, Kikuchi, D L, Willis, M, Liu, D B, Purkall, S, Sukumar, S E, Barbour, H A, Schenkein, J G, Tew   +7 more
openaire   +2 more sources