Results 201 to 210 of about 25,979 (261)

From senescence and inflammaging to systemic comorbidities: Drivers of aging‐associated periodontitis

Periodontology 2000, EarlyView.
Abstract Background Aging is accompanied by a chronic low‐grade inflammatory process, known as inflammaging, as well as immunosenescence, an age‐related decline and dysregulation of immune function, and cellular senescence, a process in which cells enter a state of irreversible growth arrest while actively releasing pro‐inflammatory factors.
James Cheng, Chew Tin Zar Aung, Samuel F. Suslavich, Sinem Esra Sahingur, Yvonne L. Hernandez‐Kapila   +4 more
wiley   +1 more source

Understanding exosomes in regenerative dentistry

Periodontology 2000, EarlyView.
Abstract Background Regenerative dentistry is shifting from cell‐based strategies to cell‐free biologics capable of orchestrating intricate tissue repair. Exosomes, nanosized extracellular vesicles carrying bioactive molecular payloads, have emerged as central modulators of intercellular communication.
Paras Ahmad, Anton Sculean, Richard J. Miron   +2 more
wiley   +1 more source

Cinnamaldehyde Protects against <i>P. gingivalis</i> Induced Intestinal Epithelial Barrier Dysfunction in IEC-6 Cells via the PI3K/Akt-Mediated NO/Nrf2 Signaling Pathway. [PDF]

Int J Mol Sci
Sampath C, Chukkapalli SS, Raju AV, Alluri LSC, Srisai D, Gangula PR.   +5 more
europepmc   +1 more source

Human β-Defensin 3 Inhibition of P. gingivalis LPS-Induced IL-1β Production by BV-2 Microglia through Suppression of Cathepsins B and L. [PDF]

Cells
Inoue E, Minatozaki S, Shimizu S, Miyamoto S, Jo M, Ni J, Tozaki-Saitoh H, Oda K, Nonaka S, Nakanishi H.   +9 more
europepmc   +1 more source

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P. gingivalis interactions with epithelial cells

Frontiers in Bioscience, 2008
Dental plaque, a microbial biofilm that accumulates on teeth and initiates periodontal disease, is composed of hundreds of different bacterial species within an organized structure. The biofilm bacteria and their byproducts irritate the gingival epithelium and induce an "inflammatory response".
Denis F, Kinane, Johnah Cortez, Galicia, Sven-Ulrik, Gorr, Panagiota Giorgios, Stathopoulou, Manjunatha, Benakanakere   +4 more
openaire   +2 more sources

Anti- P. gingivalis Response Correlates with Atherosclerosis

Journal of Dental Research, 2007
Significant associations between atherosclerosis and both Porphyromonas gingivalis, a major periodontopathogen, and the respiratory pathogen, Chlamydia pneumoniae, have been shown. Many individuals with evidence of atherosclerosis demonstrate seropositivity to these pathogens.
Ford, Pauline, Gemmell, Edward, Timms, Peter, Chan, A, Preston, F, Seymour, G   +5 more
openaire   +5 more sources

P. gingivalis drives carcinoma progression

Nature Reviews Microbiology, 2020
This study found that the oral pathogen Porphyromonas gingivalis promotes the progression of oesophageal squamous cell carcinoma.
openaire   +2 more sources

Theaflavins inhibit pathogenic properties of P. gingivalis and MMPs production in P. gingivalis-stimulated human gingival fibroblasts

Archives of Oral Biology, 2015
Theaflavins, the main polyphenols in black tea, possesses a wide range of beneficial pharmacological properties. Porphyromonas gingivalis (P. gingivalis) is a major aetiological agent associated with periodontitis, a chronic inflammatory disease affecting tooth-supporting tissues. The aim of the present study is to investigate the effect of theaflavins
Lingxue, Kong, Xia, Qi, Shiyan, Huang, Shanshan, Chen, Yafei, Wu, Lei, Zhao   +5 more
openaire   +2 more sources