Results 211 to 220 of about 25,979 (261)
Some of the next articles are maybe not open access.
Effects of Tobacco and P. gingivalis on Gingival Fibroblasts
Journal of Dental Research, 2010Cigarette smoke condensate (CSC) is the particulate matter of cigarette smoke. Porphyromonas gingivalis ( P. gingivalis) is an opportunistic pathogen involved in periodontitis. It was hypothesized that the combination of CSC and P. gingivalis would increase the collagen-degrading ability of human gingival fibroblasts (HGFs).
W, Zhang, F, Song, L J, Windsor
openaire +2 more sources
Variable Cell Responses to P. gingivalis Lipopolysaccharide
Journal of Dental Research, 2009Porphyromonas gingivalis is a major etiological agent of chronic periodontal diseases, the virulence of which has been attributed to different factors, including lipopolysaccharide (LPS). We investigated the differential responses induced by P. gingivalis LPS stimulation of human umbilical vein endothelial cells and human oral epithelial cells.
L, Kocgozlu +3 more
openaire +2 more sources
High Antibody Levels to P. gingivalis in Cardiovascular Disease
Journal of Dental Research, 2010Recent evidence suggests that strain variation in the serum IgG response to Porphyromonas gingivalis occurs in periodontal disease and cardiovascular disease (CVD). This study aimed to test the hypothesis that different P. gingivalis strains would elicit different levels of IgG, depending on a patient’s cardiovascular (CV) and periodontal health.
Bohnstedt, S. +8 more
openaire +5 more sources
[Effect of A.viscosus on growth of P. gingivalis in vitro].
Shanghai kou qiang yi xue = Shanghai journal of stomatology, 2015To investigate the effect of A. viscosus on growth of P. gingivalis in vitro.Groups as single-species(A.viscosus or P.gingivalis) and dual-species (A.viscosus and P.gingivalis) were incubated in BHI broth anaerobically at 37 degrees C, and growth curve of each group was monitored for 48 hours; Meanwhile the dual-species cultures of 2-hour interval were
Yan, Miao +3 more
openaire +2 more sources
Oral infection with P. gingivalis exacerbates autoimmune encephalomyelitis
Journal of Periodontology, 2018AbstractBackgroundOral infection of mice with P. gingivalis induces periodontal inflammation and attachment loss. The aim of the present study was to investigate whether infection of mice with P. gingivalis, exacerbates the clinical course of experimental autoimmune encephalomyelitis (EAE)—a mouse model of multiple sclerosis (MS).MethodsInduction of ...
David, Polak +3 more
openaire +2 more sources
The involvement of Kav001 in inhibition of LPS/P. gingivalis‐induced
Journal of Cellular Biochemistry, 2018AbstractTNF‐a is an important cytokine mediator of inflammation which suggests that inhibition of TNF activity may provide potential for clinical application. Recent data indicated that treatment of both human and mouse cells with Kavain significantly modulates P. gingivalis‐ and LPS‐induced TNF‐α expression.
Xiaoren Tang +6 more
openaire +2 more sources
Journal of Endotoxin Research, 2005
The fimA gene encodes the major fimbrial protein of Porphyromonas gingivalis. It has been shown to stimulate adhesion to salivary proteins and other bacteria. It is also thought to play a major role in invading and stimulating host cells. To determine whether the fimA gene represents one of the principal molecules of P.
Dana T, Graves +4 more
openaire +2 more sources
The fimA gene encodes the major fimbrial protein of Porphyromonas gingivalis. It has been shown to stimulate adhesion to salivary proteins and other bacteria. It is also thought to play a major role in invading and stimulating host cells. To determine whether the fimA gene represents one of the principal molecules of P.
Dana T, Graves +4 more
openaire +2 more sources
Archives of Oral Biology, 1992
Porphyromonas gingivalis, an important pathogen in periodontitis, produces extracellular vesicles that aggregate with Actinomyces viscosus cells. A 40-kDa outer membrane protein (OMP)-coding gene from P. gingivalis was cloned and the protein was found to be localized in these vesicles.
K, Hiratsuka +5 more
openaire +2 more sources
Porphyromonas gingivalis, an important pathogen in periodontitis, produces extracellular vesicles that aggregate with Actinomyces viscosus cells. A 40-kDa outer membrane protein (OMP)-coding gene from P. gingivalis was cloned and the protein was found to be localized in these vesicles.
K, Hiratsuka +5 more
openaire +2 more sources
Journal of Clinical Periodontology, 1994
Abstract. The present study was carried out in a private periodontal practice. 8 clinical criteria were chosen to select patients for microbiological examination. These criteria characterize prominent features of disease which at the start, during, or in the maintenance phase of treatment suggest difficulties in the progress of the periodontal therapy.
G A, Van der Weijden +5 more
openaire +2 more sources
Abstract. The present study was carried out in a private periodontal practice. 8 clinical criteria were chosen to select patients for microbiological examination. These criteria characterize prominent features of disease which at the start, during, or in the maintenance phase of treatment suggest difficulties in the progress of the periodontal therapy.
G A, Van der Weijden +5 more
openaire +2 more sources
Distinctive pathways characterize A. actinomycetemcomitans and P. gingivalis.
Molecular biology reports, 2016The study aimed to compare the molecular mechanism of Porphuromonas gingivalis (P. gingivalis) and Aggregatibacter actinomycetemcomitans (A. actinomycetemcomitans). With microarray dataset (GSE9723) from Gene Expression Omnibus, differentially expressed genes (DEGs) were identified comparing normal cell samples with A.
Jing, Lv +3 more
openaire +1 more source