Results 21 to 30 of about 460,858 (405)

Src Dependent Pancreatic Acinar Injury Can Be Initiated Independent of an Increase in Cytosolic Calcium [PDF]

open access: yes, 2013
Several deleterious intra-acinar phenomena are simultaneously triggered on initiating acute pancreatitis. These culminate in acinar injury or inflammatory mediator generation in vitro and parenchymal damage in vivo. Supraphysiologic caerulein is one such
A Gnoni   +72 more
core   +8 more sources

Ultrasound-Assisted versus Endoscopic Nasojejunal Tube Placement for Acute Pancreatitis: A Retrospective Feasibility Study

open access: yesGastroenterology Research and Practice, 2021
Objective. The optimal technique for nasojejunal tube (NJT) placement in terms of facilitating early enteral nutrition (EN) in patients with acute pancreatitis (AP) is unclear. In this study, we aimed to evaluate the impact of two common techniques on EN
Gang Li   +6 more
doaj   +1 more source

Plasma metagenomic next-generation sequencing of microbial cell-free DNA detects pathogens in patients with suspected infected pancreatic necrosis

open access: yesBMC Infectious Diseases, 2022
Background Infected pancreatic necrosis (IPN) is a life-threatening complication of acute pancreatitis (AP). Timely diagnosis of IPN could facilitate appropriate treatment, but there is a lack of reliable non-invasive screening tests.
Donghuang Hong   +10 more
doaj   +1 more source

Pathophysiology of acute experimental pancreatitis: Lessons from genetically engineered animal models and new molecular approaches [PDF]

open access: yes, 2005
The incidence of acute pancreatitis is growing and worldwide population-based studies report a doubling or tripling since the 1970s. 25% of acute pancreatitis are severe and associated with histological changes of necrotizing pancreatitis. There is still
Anne Barbara Tietz   +38 more
core   +1 more source

Primary hyperparathyroidism can generate recurrent pancreatitis and secondary diabetes mellitus – A case report [PDF]

open access: yes, 2019
Introduction. Acute or recurrent pancreatitis may be a complication of primary hyperparathyroidism and patients with previous episodes of pancreatitis may develop secondary diabetes mellitus. Case report.
Dănciulescu Miulescu, Rucsandra Elena   +4 more
core   +2 more sources

A Suspicious Pancreatic Mass in Chronic Pancreatitis: Pancreatic Actinomycosis [PDF]

open access: yesCase Reports in Oncological Medicine, 2015
Introduction. Pancreatic actinomycosis is a chronic infection of the pancreas caused by the suppurative Gram-positive bacteriumActinomyces. It has mostly been described in patients following repeated main pancreatic duct stenting in the context of chronic pancreatitis or following pancreatic surgery.
P. Laukens   +6 more
openaire   +4 more sources

Pancreatitis following Olanzapine Therapy: A Report of Three Cases [PDF]

open access: yes, 2007
CONTEXT: Atypical antipsychotic agents (clozapine, olanzapine) have been linked to metabolic effects and acute pancreatitis. CASE REPORT: We reviewed the inpatient and outpatient records of three patients who developed acute pancreatitis while being ...
Azar, Riad   +3 more
core   +4 more sources

ACG Clinical Guideline: Chronic Pancreatitis.

open access: yesAmerican Journal of Gastroenterology, 2020
Chronic pancreatitis (CP) is historically defined as an irreversible inflammatory condition of the pancreas leading to varying degrees of exocrine and endocrine dysfunction.
T. Gardner   +5 more
semanticscholar   +1 more source

Incidence and risk factors of nasogastric feeding intolerance in moderately-severe to severe acute pancreatitis

open access: yesBMC Gastroenterology, 2022
Background The importance of enteral nutrition (EN) in acute pancreatitis (AP) has been emphasised. Nasogastric (NG) feeding has been the preferred route for EN delivery in most AP patients intolerant to oral intake. However, gastric feeding intolerance (
Jiajia Lin   +9 more
doaj   +1 more source

Essential Role of Lyn in Fibrosis. [PDF]

open access: yes, 2016
Fibrotic disorders involve replacement of normal parenchyma with myofibroblasts, which deposit connective tissue, leading to obliteration of the function of the underlying organ.
Birtolo, Chiara   +10 more
core   +2 more sources

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