Correction: Peptide-based antimicrobial effect against carbapenem-resistant <i>Acinetobacter baumannii</i>: preclinical drug assessment and translational potential. [PDF]
Lee HJ +6 more
europepmc +1 more source
Parkes observations for project P1054 semester 2025OCTS_06
Burgay, Marta +32 more
openalex +1 more source
E2A selectively regulates TGF‐β–induced apoptosis in KRAS‐mutant non‐small cell lung cancer
Ability to induce apoptosis by TGF‐β is frequently lost in advanced lung adenocarcinoma despite intact TGF‐β signaling. We identify E2A as a mutant KRAS–dependent mediator of resistance to TGF‐β–induced apoptosis. TGF‐β induces E2A via SMAD3 in mutant KRAS cells, and E2A silencing restores apoptosis and enhances radiation response in cell lines ...
Sergei Chuikov +3 more
wiley +1 more source
Renal Nonenhancement From Aortocaval Fistula in Abdominal Aortic Aneurysm. [PDF]
Bok Y, Park JH.
europepmc +1 more source
New Hyde Park-Garden City Park Union Free School District and New Hyde Park-Garden City Park Custodial and Transportation Unit, CSEA, Local 1000 AFSCME, AFL-CIO (2009) [PDF]
core +1 more source
Beyond its role in immune evasion, this study identified that CD47 drives tumor‐intrinsic signaling in non‐small cell lung cancer (NSCLC). Transcriptomic profiling and functional studies revealed that CD47 regulates cell adhesion, migration, and metastasis through an ERK–EMT signaling axis.
Asa P.Y. Lau +8 more
wiley +1 more source
Adapting a systematic conservation planning tool for supporting accessible and diverse urban greenspace recreation. [PDF]
Huang J, Gergel SE, McHale MR.
europepmc +1 more source
KDM7A and KDM1A inhibition suppresses tumour promoting pathways in prostate cancer
Treatment resistance is a major challenge for patients with advanced prostate cancer. This study examined an alternative approach to target the major prostate cancer‐promoting pathway by targeting epigenetic factors, whose levels are higher in tumours.
Jennie N Jeyapalan +16 more
wiley +1 more source
We analyze cisplatin–DNA adducts (CDAs) and double‐strand breaks (DSBs) in a cell‐cycle‐dependent manner. We find that CDAs form similarly across all cell cycle phases. DSBs arise only in S‐phase. CDAs might not directly impair DSB repair, but S‐phase DSB lesions evolve in the presence of CDAs and disrupt repair in G2, also causing radiosensitization ...
Ye Qiu +10 more
wiley +1 more source

