Results 231 to 240 of about 2,493,525 (282)

Novel Use of Intravascular Lithotripsy for the Mobilization of an Entrapped Coronary Guidewire in a Heavily Calcified LAD/DB Bifurcation Lesion—A Case Report

Catheterization and Cardiovascular Interventions, EarlyView.
ABSTRACT A 64‑year‑old man with a Non–ST‑elevation myocardial infarction (NSTEMI) underwent coronary angiography, which revealed single‑vessel disease with a high‑grade proximal and a severely calcified mid‑LAD lesion just proximal to a diagonal branch (DB). Both the LAD and the DB were wired.
Hansmartin Jetter, Jochen Genzel, Wolfram Voelker   +2 more
wiley   +1 more source

Early and short-term use of proprotein convertase anti-subtilisin-kexin type 9 inhibitors on coronary plaque stability in acute coronary syndrome. [PDF]

Eur Heart J Open
Uehara H, Kajiya T, Abe M, Nakata M, Hosogi S, Ueda S.   +5 more
europepmc   +1 more source

Incidence of Periprocedural Myocardial Injury and Infarction After Intravascular Lithotripsy for Calcified Coronary Artery Stenosis

Catheterization and Cardiovascular Interventions, EarlyView.
ABSTRACT Background Intravascular lithotripsy (IVL) has emerged as an effective technique for modifying heavily calcified coronary plaques, facilitating optimal stent expansion while minimizing procedural complications. However, data on periprocedural myocardial injury (PMI) and infarction (PPMI) following IVL remain scarce.
Gwaenaelle Aellen, Dorian Garin, Yannick Faucherre, Serban Puricel, Mario Togni, Stéphane Cook   +5 more
wiley   +1 more source

Spatial Mismatch Between Angiographic Obstruction and Plaque Destabilization in Acute Coronary Syndromes: A Conceptual Narrative Review

Catheterization and Cardiovascular Interventions, EarlyView.
ABSTRACT The identification of the “culprit lesion” in acute coronary syndromes (ACS) traditionally relies on coronary angiography, assuming that the most severe stenosis, abrupt occlusion, or largest filling defect marks the site of plaque destabilization.
Fernando Quevedo‐Candela, Olivier F. Bertrand, Pedro Cepas‐Guillén, Jean‐Pierre Déry, David García‐Labbé, Andrés Ruhl, Eduardo Flores‐Umanzor, Tomas Cieza   +7 more
wiley   +1 more source

Treatment of advanced atherosclerotic mice with ABT-263 reduced indices of plaque stability and increased mortality. [PDF]

JCI Insight
Karnewar S, Karnewar V, Shankman LS, Owens GK.   +3 more
europepmc   +1 more source

4′‐ThioRNA Substitution and Sense‐Strand Segmentation for the Design of Functional Fully Modified siRNAs Targeting SARS‐CoV‐2

Chemistry – A European Journal, EarlyView.
4′‐ThioRNA was broadly tolerated across multiple positions in SARS‐CoV‐2‐targeting siRNAs, but full 4'‐thio modification of both strands caused a reduction in RNAi activity. Sense‐strand segmentation improved RNAi and antiviral activity, particularly when the nick was located at position 10 from the 5′‐end of the sense strand, thereby unlocking the ...
Yuhei Nogi, Jun Tsukimoto, Noriko Saito‐Tarashima, Takaaki Koma, Masako Nomaguchi, Noriaki Minakawa   +5 more
wiley   +1 more source

miR-449a disturbs atherosclerotic plaque stability in streptozotocin and high-fat diet-induced diabetic mice by targeting CEACAM1. [PDF]

Diabetol Metab Syndr
Yu J, Liu H, Chen Y, Wang L, Chen P, Zhao Y, Ou C, Chen W, Hu J, Wang Y, Wang Y.   +10 more
europepmc   +1 more source

Some of the next articles are maybe not open access.

Related searches:

Statins and Plaque Stability

European Journal of Cardiovascular Prevention & Rehabilitation, 2003
3-hydroxy-3-methylglutaryl coenzyme A (HMG-CoA) reductase inhibitors are now the standard of care for patients with hypercholesterolaemia. This class of inhibitors, known as 'statins', has been shown to reduce cardiovascular morbidity and mortality. Accumulating data demonstrates a variety of mechanisms in which HMG-CoA reductase inhibition benefits ...
Mardi, Gomberg-Maitland, Valentin, Fuster, Zahi Adel, Fayad, Juan Jose, Badimon, Roberto, Corti   +4 more
openaire   +2 more sources

Mechanisms of plaque stabilization with statins

The American Journal of Cardiology, 2003
The traditional view of cardiovascular disease held that the degree of stenosis defined high-risk lesions and that removal of cholesterol shrank these lesions and thereby enlarged the lumen. Advances in understanding of the pathophysiology of the acute coronary syndromes refute this view.
Peter, Libby, Masanori, Aikawa
openaire   +2 more sources

Macrophages and atherosclerotic plaque stability

Current Opinion in Lipidology, 1996
Physical disruption of atheroma frequently causes coronary thrombosis. Ruptured plaques usually have thin fibrous caps overlying a large thrombogenic lipid core rich in lipid-laden macrophages. The biology of plaque monocyte-derived macrophages thus assumes critical importance in understanding plaque instability.
P, Libby, Y J, Geng, M, Aikawa, U, Schoenbeck, F, Mach, S K, Clinton, G K, Sukhova, R T, Lee   +7 more
openaire   +2 more sources