Results 171 to 180 of about 18,594 (231)

Plasmodium berghei: Immunosuppression and hyperimmunoglobulinemia

Experimental Parasitology, 1977
Abstract Fatal Plasmodium berghei infection in BALB c mice caused permanent and complete depression of the mice's immune responsiveness to sheep erythrocytes beginning in the period of the first peak parasitemia and lasting until all the animals had died.
L G, Poels, C C, van Niekerk
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Disk Electrophoresis of Plasmodium berghei

The Journal of Parasitology, 1966
A method of analytic separation of soluble proteins in aqueous extracts of Plasmodium berghei is described. Extracts are prepared by sonication of suspensions of whole washed parasites obtained by saponin lysis of infected rat red cells. Separation of the extracts into reproducible electrophoretic patterns is carried out by zone electrophoresis on ...
W A, Sodeman, H E, Meuwissen
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Metabolism of Plasmodium berghei

Experimental Parasitology, 1968
The free parasites of Plasmodium berghei, obtained from infected cells of rats using an antiserum method, were investigated to study the operation of Krebs cycle. P.
Nagarajan, K
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Enzyme variation in Plasmodium berghei

Transactions of the Royal Society of Tropical Medicine and Hygiene, 1970
Abstract 10 strains of P. berghei representing 4 subspecies—P.b. berghei, P.b. yoelii, P.b. killicki and a “P. berghei-like” parasite from Nigeria were used in this study. The electrophoretic band patterns of 4 enzymes—glucose phosphate isomerase, adenylate kinase, 6-phosphogluconate dehydrogenase and malate dehydrogenase were examined in all strains.
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Chloroquine Resistance in Plasmodium Berghei

The American Journal of Tropical Medicine and Hygiene, 1966
Summary A strain of Plasmodium berghei was passed through mice which were maintained on a diet containing 0.008% chloroquine diphosphate and which had been treated intravenously with ethyl palmitate to destroy or diminish the phagocytic functions of the spleen.
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Plasmodium berghei and Plasmodium knowlesi: Serum binding to sporozoites

Experimental Parasitology, 1980
Abstract Plasmodium berghei salivary gland and oocyst sporozoites were examined with fluorescein isothiocyanate (FITC)-lectins to determine if sporozoites had carbohydrate-containing molecules on their surfaces. None of the eight fluorescein isothiocyanate-lectins bound to the sporozoites.
S, Schulman, J D, Oppenheim, J P, Vanderberg   +2 more
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Exotoxin in malaria infection (Plasmodium berghei)

Experientia, 1967
Swiss Mause, die unbehandelt eine Infektion mitPl. berghei niemals uberleben, wurden mit Serum von frisch infizierten Tieren (12. Infektionstag) immunisiert und anschliessend infiziert. Obgleich sich danach in allen Fallen eine schwere Parasitamie entwickelte, heilten 44% derjenigen Tiere nach dem 35.
S, Jerusalem, D, Bruchhausen
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Plasmodium berghei: Oxidant defense system

Experimental Parasitology, 1985
Glutathione oxidant defense system protects the erythrocyte from oxidative damage. This defense system was studied in mouse erythrocytes infected with Plasmodium berghei and in isolated parasites. The efficiency of this system was found to be increased in parasitized erythrocytes compared to the normal erythrocytes.
R K, Seth, A S, Saini, T S, Jaswal
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Pathophysiology of Plasmodium berghei infection in mice

Experimental Parasitology, 1965
Abstract Significant biochemical changes were detected in the serum of mice infected with P. berghei . Marked increases in SGP-T and SGO-T transaminases were observed as early as 2 days after infection. Lower fasting glucose levels occurred in heavily infected mice 4 days after infection.
E H, Adun, J S, Williams, F C, Meroney, G, Hutt   +3 more
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Interferon Induced by Plasmodium berghei

Science, 1968
An inhibitor of virus is demonstrable in the serums of mice infected with Plasmodium berghei . The titer of the inhibitor, detectable within 10 hours after injection of parasitized blood, increases rapidly until 30 to 40 hours when it levels off or decreases slightly before reaching a plateau.
K Y, Huang, W W, Schultz, F B, Gordon
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