Plasmalogenic Lipid Analogs as Platelet-Activating Factor Antagonists: A Potential Novel Class of Anti-inflammatory Compounds. [PDF]
Rong P+4 more
europepmc +1 more source
Metabolic, membrane, and functional responses of human polymorphonuclear leukocytes to platelet-activating factor [PDF]
LM Ingraham+5 more
openalex +1 more source
A multi‐functional shear‐thinning hydrogel is engineered to treat enterocutaneous fistulas. This hydrogel can resist external pressures allowing it to remain within the fistula tract and is shown to have significant antimicrobial, pro‐healing, and anti‐inflammatory properties, which were validated in in vitro and in vivo experiments.
Jinjoo Kim+6 more
wiley +1 more source
Intoxicated thermal burn injury-mediated systemic immunosuppression involves platelet-activating factor and microvesicle particles. [PDF]
Lohade RP+3 more
europepmc +1 more source
Engineering the Immune Response to Biomaterials
This paper reviews immune responses to biomaterials, stages of evolution in relation to managing these responses ranging from preventing to modulating them. It also discusses state‐of‐the‐art strategies to modulate these responses and modes of their delivery.
Abolfazl Salehi Moghaddam+12 more
wiley +1 more source
Evidence that keratinocyte microvesicle particles carrying platelet-activating factor mediate the widespread multiorgan damage associated with intoxicated thermal burn injury. [PDF]
Lohade RP+10 more
europepmc +1 more source
The Latest Advances in Mechanically Robust Self‐Healing Polyurea Based on Dynamic Chemistry
Solving the contradiction between strength, toughness, and self‐healing properties is extremely important to realize the breakthrough of polyurea applications. This review summarizes and discusses common strategies for the preparation of high‐performance and self‐healing polyurea materials based on dynamic chemistry, providing molecular‐level insights ...
Yujia Hao, Guangming Zhu
wiley +1 more source
Transcriptome Landscape of Cancer‐Associated Fibroblasts in Human PDAC
AP‐1 family members FOS and JUN regulate the malignant phenotype conversion of normal fibroblasts (NFs) to normal‐like cancer‐associated fibroblasts (nCAFs), while transforming growth factor‐β (TGFβ) and interferon‐γ (IFNγ) signals triggers the interconversion between classic myofibroblastic CAFs (myCAFs) and inflammatory CAFs (iCAFs), respectively ...
Mengyu Tao+15 more
wiley +1 more source