Results 161 to 170 of about 1,069 (193)

Polioencephalomalacia of Dogs with Thiamine Deficiency

Veterinary Pathology, 1977
A naturally occurring neurological disease occurred in six dogs fed cooked meat. Clinical signs were anorexia, progressive spastic paraparesis, recumbency, convulsions and death. The disease was characterized by bilaterally symmetrical spongy change and necrosis of brainstem nuclei with a lesion distribution pattern similar to that in thiamine ...
D H, Read, R D, Jolly, M R, Alley
openaire   +2 more sources

POLIOENCEPHALOMALACIA-LIKE DISEASE IN PRONGHORNS (ANTILOCAPRA AMERICANA)

Journal of Wildlife Diseases, 1983
A disease resembling thiamine-responsive polioencephalomalacia of domestic ruminants is described in four wild pronghorn from Saskatchewan. One animal was found dead, two were recumbent and unable to rise and the fourth was staggering and ataxic. Lesions were confined to the brain and consisted of brain swelling with herniation, symmetrical hemorrhagic
G, Wobeser, P Y, Daoust, H M, Hunt
openaire   +2 more sources

Ovine Polioencephalomalacia Associated With Dietary Sulphur Intake

Journal of Veterinary Medicine Series A, 1991
SummaryFifty‐six female crossbred two‐month‐old lambs were housed in individual cages, and fed a basic ration of barley (59%), soybean meal (5%), and alfalfa (32%) prepared to meet NRC nutrient requirements. Four percent of the diet contained a standard salt mix to which the factors inorganic sulphur (S) and thiamine (B1) were added.
C G, Rousseaux, A A, Olkowski, A, Chauvet, S R, Gooneratne, D A, Christenson   +4 more
openaire   +2 more sources

Polioencephalomalacia in Captive Harbour Seals (Phoca vitulina)

Journal of Veterinary Medicine Series A, 2003
SummaryIn a colony of 11 harbour seals (Phoca vitulina Linné 1758) two episodes of central nervous disorders occurred within 2 years causing fatalities in seven adult animals. Clinical signs comprised dyspnoea, anorexia, apathy, incoordination and lateral recumbency. Vitamin B complex therapy was successful once. Pathomorphological examination of seven
P, Wohlsein, M, Peters, F, Geburek, F, Seeliger, M, Böer   +4 more
openaire   +2 more sources

Polioencephalomalacia in a juvenile hooded seal ( Cystophora cristata )

Veterinary Record, 2006
appeared grossly normal. No significant bacteria were recovered on microbiology and all the examinations for Brucella species were negative. Histological examination of coronal slices of the fore-, midand hindbrain and one sagittal slice of the cerebellum showed widespread lesions of polioencephalomalacia throughout the cerebrum.
Dagleish, MP, Patterson, IAP, Foster, G, Reid, RJ, Brain, LTA, Buxton, D   +5 more
openaire   +3 more sources

Cerebrocortical necrosis (Polioencephalomalacia) in a goat

New Zealand Veterinary Journal, 1963
Abstract Extract A Saanen goat, approximately two years old, which had been tethered on clean pasture, showed sudden onset of blindness, collapse, and aimless paddling movements of the legs when stimulated by touch. Respirations were deep and irregular. The temperature was normal. Death ensued 48 hours after the onset of symptoms.
B.W. Manktelow, W.T. Reid
openaire   +1 more source

Relationship between sulfur, thiamine and polioencephalomalacia

The Bovine Practitioner, 2002
Thiamine deficiency has been classically described as the cause of ruminant polioencephalomalacia (PEM). More recently excess dietary sulfur has been shown to be a major cause of PEM. This paper reviews the relationship between PEM and thiamine metabolism in mature cattle, thiaminase in plants, rumen acidosis and excess dietary sulfur.
Niles, G. A., Morgan, S. E., Edwards, W. C.   +2 more
openaire   +1 more source

Sulfur-induced polioencephalomalacia in stocker calves.

Veterinary and human toxicology, 2001
Calves from 3 farms exhibited blindness, head pressing, and circling before death. Brain lesions confirmed polioencephalomalacia. Excess sulfur was found in the diets on all 3 farms in corn by-products or molasses based supplements. Corn gluten feed and corn steep liquor (by-products of the refinement of corn for ethanol), corn syrup, corn gluten, corn
G A, Niles, S E, Morgan, W C, Edwards
openaire   +1 more source

Polioencephalomalacia in Feedlot Lambs

Journal of the American Veterinary Medical Association, 1975
R E, Pierson, R, Jensen
openaire   +2 more sources

Polioencephalomalacia in a llama.

The Canadian veterinary journal = La revue veterinaire canadienne, 2008
A 5-year-old, female llama (Lama glama) developed acute, progressive neurological disease, characterized by recumbency, muscle fasciculations, intermittent convulsions/opisthotonos, and absent menace responses. Postmortem histopathologic lesions, limited to the cerebral cortex, consisted of necrosis of the superficial and deep laminae.
openaire   +1 more source