Results 71 to 80 of about 23,979 (222)

Idiopathic inflammatory myopathies: Contributions from India

open access: yesIndian Journal of Rheumatology, 2020
A careful literature search would reveal significant contributions by Indian clinicians and basic researchers in the field of Idiopathic inflammatory myopathies (IIMs).
Anand Narayan Malaviya
doaj   +1 more source

Rare Gain‐of‐Function Variants in IDH1 and IDH2 Confer Increased Risk of Immune‐Mediated Rheumatic Diseases

open access: yesACR Open Rheumatology, Volume 8, Issue 4, April 2026.
Objective Gain‐of‐function variants in IDH1 and IDH2 are enriched among individuals with coexisting myeloid neoplasms and autoimmune diseases. However, the association between IDH1/IDH2 variants and rheumatic diseases independent of hematologic neoplasms remains unclear. We investigated the association between rare gain‐of‐function and loss‐of‐function
Mahmut S. Kaymakci   +3 more
wiley   +1 more source

Acute Onset Polymyositis after Prolactinoma Extirpation

open access: yesCase Reports in Rheumatology, 2014
Hyperprolactinemia has been related to autoimmune diseases. Herein, we describe a case of a female with a prolactin producer pituitary macroadenoma who developed severe polymyositis one month after its removal. The patient had very high levels of CPK and
Juan Jakez-Ocampo   +2 more
doaj   +1 more source

Hypercapnic respiratory failure during pregnancy due to polymyositis-related respiratory muscle weakness: a case report

open access: yesJournal of Medical Case Reports, 2017
Background Polymyositis is a rare medical disorder complicating pregnancy. Ventilatory muscle weakness leading to respiratory failure is an uncommon manifestation of this autoimmune disease.
Husain Shabbir Ali   +3 more
doaj   +1 more source

Brain Involvement in Leishmaniasis

open access: yesCell Biochemistry and Function, Volume 44, Issue 4, April 2026.
ABSTRACT Leishmaniasis is a neglected tropical disease caused by infection with the protozoan parasite Leishmania and it is a significant global health problem. The disease has a wide clinical spectrum, from tegumentary leishmaniasis (TL) that encompasses cutaneous (CL), mucosal (ML) and cutaneous‐diffuse (CDL) forms, to the potentially fatal systemic ...
Camila S. Freitas   +2 more
wiley   +1 more source

Polymyositis [PDF]

open access: yesBMJ, 2012
Paul, Truepenny   +2 more
openaire   +3 more sources

An electrophysiological investigation of skeletal muscles in polymyositis Investigação eletrofisiológica de músculos esqueléticos na polimíosite

open access: yesArquivos de Neuro-Psiquiatria, 1976
An electrophysiological study has been made of the extensor digitorum brevis, thenar and hypothenar muscles in 25 patients with chronic and acute polymyositis.
R. E. P. Sica   +3 more
doaj  

Polymyositis and dermatomyositis – challenges in diagnosis and management

open access: yesJournal of Translational Autoimmunity, 2019
Polymyositis (PM) and dermatomyositis (DM) are different disease subtypes of idiopathic inflammatory myopathies (IIMs). The main clinical features of PM and DM include progressive symmetric, predominantly proximal muscle weakness.
Shu-Han Yang   +2 more
doaj   +1 more source

Giant cell myocarditis masquerading as orbital myositis with a rapid, fulminant course necessitating mechanical support and heart transplantation. [PDF]

open access: yes, 2017
Giant cell myocarditis (GCM), a rapidly progressive inflammation of the myocardium, is associated with fulminant heart failure, refractory ventricular arrhythmias, and conduction system abnormalities.
Aksoy, Olcay   +5 more
core   +1 more source

A New Immunofluorescence Assay Allows the Sensitive Detection of Anti‐Cytosolic 5′‐Nucleotidase 1A Autoantibodies

open access: yesEuropean Journal of Immunology, Volume 56, Issue 4, April 2026.
Anti‐cN1A autoantibodies are helpful for diagnosing inclusion body myositis (IBM). We have developed an immunofluorescence assay based on the perinuclear accumulation of GFP‐cN1A. The analysis of patient sera indicated that this assay shows a higher sensitivity for anti‐cN1A in IBM compared to alternative assays, without loss of specificity.
Fleur N. Brinkman   +3 more
wiley   +1 more source

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