Results 181 to 190 of about 36,847 (227)
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Annual Review of Virology, 2016
Mammalian polyomaviruses are characterized by establishing persistent infections in healthy hosts and generally causing clinical disease only in hosts whose immune systems are compromised. Despite the fact that these viruses were discovered decades ago, our knowledge of the mechanisms that govern viral persistence and reactivation is limited.
Michael J, Imperiale, Mengxi, Jiang
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Mammalian polyomaviruses are characterized by establishing persistent infections in healthy hosts and generally causing clinical disease only in hosts whose immune systems are compromised. Despite the fact that these viruses were discovered decades ago, our knowledge of the mechanisms that govern viral persistence and reactivation is limited.
Michael J, Imperiale, Mengxi, Jiang
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Polyomavirus inactivation – A review
Biologicals, 2013Polyomavirus inactivation has been studied since the 1950s when it became apparent that certain polio vaccines were contaminated with SV40. Relatively high temperatures (≥70 °C) are required to effect thermal inactivation of the polyomaviruses. The chemical inactivants that are effective (β-propiolactone, ethanol, sodium hydroxide, and formaldehyde ...
Raymond W, Nims, Mark, Plavsic
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The biology of JC polyomavirus
Biological Chemistry, 2017Abstract JC polyomavirus (JCPyV) is the causative agent of a fatal central nervous system demyelinating disease known as progressive multifocal leukoencephalopathy (PML). PML occurs in people with underlying immunodeficiency or in individuals being treated with potent immunomodulatory therapies.
Benedetta Assetta, Walter J. Atwood
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Transplantation Proceedings, 2003
Because it is an important factor affecting renal transplant function, BK infections are significant problem in posttransplant. BK nephropathy develops in 5% of renal allograft recipients, in most cases within the first year after the procedure. The gold standard for BK nephropathy diagnosis is still immunohistochemical staining for large T antigen in ...
B, Matłosz +11 more
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Because it is an important factor affecting renal transplant function, BK infections are significant problem in posttransplant. BK nephropathy develops in 5% of renal allograft recipients, in most cases within the first year after the procedure. The gold standard for BK nephropathy diagnosis is still immunohistochemical staining for large T antigen in ...
B, Matłosz +11 more
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A possible bovine polyomavirus
Archives of Virology, 1980A virus with the characteristics of a polyomavirus was isolated from tissue cultures prepared from the kidneys of a healthy new-born calf.
W, Coackley, D, Maker, V W, Smith
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Polyomavirus nephropathy in kidney transplantation
Progress in Transplantation, 2004Polyomavirus nephropathy has become an important complication in kidney transplantation, with a prevalence of 1% to 8%. Unfortunately, the risk factors for polyomavirus nephropathy and renal allograft loss are not well defined. The definitive diagnosis is made through assessment of a kidney transplant biopsy.
Jennifer, Trofe +7 more
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Polyomavirus Latency and Human Tumors
The Journal of Infectious Diseases, 1994---
DE MATTEI, Monica +5 more
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Infection with polyomavirus JC
BMJ, 2011Is highly prevalent, and can be fatal in immunocompromised people The recent discovery of new neurological syndromes that result from neuronal infection with polyomavirus JC, also known as JC virus, and the presence of this virus in the grey matter are currently under debate.1 It has been suggested that JC virus is associated with cognitive decline ...
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New polyomavirus species identified in nutria, Myocastor coypus polyomavirus 1
Archives of Virology, 2018A novel polyomavirus (PyVs) comprising 5,422 bp was identified by high-throughput sequencing (HTS) in pooled organs of nutria (Myocastor coypus). The new genome displays the archetypal organization of PyVs, which includes open reading frames for the regulatory proteins small T antigen (sTAg) and large T antigen (LTAg), as well as for the capsid ...
M S, da Silva +10 more
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1995
The hamster polyomavirus (HaPV) was originally described in 1967 by Graffi et al. as a virus associated with skin epithelioma of the Syrian hamster.(1–4) The tumors appear spontaneously in animals at about 3 months to more than 1 year of age in a laboratory colony bred in Berlin Buch, Germany (HaB).
Siegfried Scherneck, Jean Feunteun
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The hamster polyomavirus (HaPV) was originally described in 1967 by Graffi et al. as a virus associated with skin epithelioma of the Syrian hamster.(1–4) The tumors appear spontaneously in animals at about 3 months to more than 1 year of age in a laboratory colony bred in Berlin Buch, Germany (HaB).
Siegfried Scherneck, Jean Feunteun
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