Results 111 to 120 of about 998 (161)

A Sclerodermiform Porphyria Cutanea Tarda

Internal Medicine, 2010
Amezyane, Taoufik, Abouzahir, Ali, Fatihi, Jamal, Sekkach, Youssef, Mahassin, Fatima, Sedrati, Omar, Ghafir, Driss, Ohayon, Victor   +7 more
openaire   +3 more sources

Porphyria Cutanea Tarda and Hyperkeratosis

International Journal of Clinical Practice, 1972
openaire   +2 more sources

Porphyria cutanea tarda [PDF]

MMW - Fortschritte der Medizin, 2012
openaire   +1 more source

A rare case of porphyria cutanea tarda in a patient with a homozygous hereditary hemochromatosis gene H63D mutation in the setting of hereditary hemochromatosis

JAAD Case Reports
Jonathan Banta, MD, Joshua Collins, DO, Todd Kobayashi, MD   +2 more
doaj   +1 more source

Paraneoplastic porphyria cutanea tarda associated with cholangiocarcinoma: Case report

The Turkish Journal of Gastroenterology, 2007
Mehmet SÖKMEN, Hüseyin DEMİRSOY, Özdal ERSOY, Gonca GÖKDEMİR, Nihat AKBAYIR, Çetin KARACA, Kamil ÖZDİL, Beşir KESİCİ, Can ÇALIŞKAN, Banu YILMAZ   +9 more
doaj  

Porphyria cutanea tarda

Australasian Journal of Dermatology, 2000
SUMMARYPorphyria cutanea tarda (PCT) is a metabolic disorder of haem biosynthesis caused by decreased activity of uroporphyrinogen decarboxylase. Porphyria cutanea tarda is manifest by fragility, erosions, bullae, milia and scars on sun‐exposed skin.
N R, Bleasel, G A, Varigos
core   +7 more sources

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Therapy of porphyria cutanea tarda

Expert Opinion on Pharmacotherapy, 2005
Porphyria cutanea tarda (PCT) is the most common type of porphyria. There is an association of PCT with haemochromatosis, diabetes mellitus and hepatitis C infection. The basis of treatment of PCT consists of three elements: avoidance of triggering factors, iron depletion and porphyrin elimination.
Erich Köstler
exaly   +3 more sources

Porphyria Cutanea Tarda

Seminars in Liver Disease, 1998
Porphyria cutanea tarda (PCT) is a skin disease that results from decreased activity of uroporphyrinogen decarboxylase (UROD). About 80% of patients have the sporadic (type I) form in which UROD deficiency is restricted to the liver. Others have familial (type II) PCT in which mutations in the UROD gene are inherited in an autosomal dominant pattern ...
C, Fritsch, S, von Schmiedeberg, P, Lehmann   +2 more
  +7 more sources

HLA and porphyria cutanea tarda

Archives of Dermatological Research, 1980
R Enriquez De Salamanca
exaly   +3 more sources

Porphyria cutanea tarda and Pregnancy

Dermatologica, 2009
A female patient with porphyria cutanea tarda (PCT) clinically cured by a low-dose chloroquine treatment showed neither exacerbation of the disease nor an increased excretion of urinary porphyrins during pregnancy. The role of estrogens and gestagens as clinical manifestation factors of PCT is discussed.
G, Goerz, G, Hammer
openaire   +2 more sources