Results 141 to 150 of about 23,339 (176)
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FAMILIAL PORPHYRIA CUTANEA TARDA *
Australasian Journal of Dermatology, 1985SUMMARYThe results of erythrocyte uroporphyrinogen decarboxylase estimations of members of the family of a child with porphyria cutanea tarda are reported.
M, Rogers, V, Poulos
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Porphyria Cutanea Tarda and Pregnancy
Obstetrical & Gynecological Survey, 1996Porphyria cutanea tarda is the most common disorder of porphyrin metabolism in Europe and North America. The disorder is characterized by specific cutaneous lesions, associated systemic findings, and excessive accumulation and excretion of uroporphyrin and coproporphyrins. Reports of this condition associated with pregnancy are scarce in the literature.
J R, Loret de Mola +2 more
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Porphyria cutanea tarda and sarcoidosis
Journal of the American Academy of Dermatology, 1980A 38-year-old black woman presented with a multisystem disease characterized by malaise, fever, sweats, and diffuse hyperpigmentation. Laboratory examinations showed anemia, elevated alkaline phosphatase, granulomas in the liver and bone marrow, and elevated porphyrins in the urine and feces, characteristic of porphyria cutanea tarda (PCT).
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THE LIVER IN PORPHYRIA CUTANEA TARDA
Annals of Internal Medicine, 1960Excerpt The porphyrias belong to the group of diseases that have been called inborn errors of metabolism.
J, WALDENSTROM, B, HAEGER
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Porphyria Cutanea Tarda and Estrogens
JAMA: The Journal of the American Medical Association, 1967Porphyria cutanea tarda can be induced by a variety of compounds, including estrogens. Cutaneous manifestations of the disease are characterized chiefly by a light- and trauma-sensitive eruption of exposed areas. In two cases occurring secondary to estrogens, improvement followed estrogen withdrawal.
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Porphyria cutanea tarda in Ethiopia
Transactions of the Royal Society of Tropical Medicine and Hygiene, 1981The clinical, biochemical and histological features of 75 Ethiopians with porphyria cutanea tarda (PCT) are described. PCT in Ethiopia is definitely related to alcohol abuse and there is no clinical evidence for hereditary predisposition. Significant elevation of transaminases and bromsulphthalein retention, moderate to marked hepatic siderosis and ...
E, Tsega +3 more
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The inheritance of porphyria cutanea tarda
International Journal of Biochemistry, 1980Abstract The porphyrin excretion of 203 blood relatives of 46 PCT patients was analyzed. 48 relatives (23.64%) showed abnormalities of porphyrin pattern and 19 of them suffered overt PCT. On the contrary, in some families no one relative showed any clinical or biochemical abnormality. These data confirm the existence of two forms of PCT: the sporadic
R, Enríquez de Salamanca +5 more
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OBSERVATIONS ON PORPHYRIA CUTANEA TARDA
Archives of Dermatology, 1954PORPHYRIA is a relatively rare, inherited fault of pyrrole metabolism which produces a wide variety of clinical symptoms from time to time or remains latent for years or for life. It is characterized by the excretion of abnormal kinds and amounts of porphyrins, chiefly uroporphyrin and its precursors.
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