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How We Treat Primary Hyperoxaluria Type 1. [PDF]
Clin J Am Soc NephrolBreeggemann MC +4 more
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Correction: Lumasiran: A Review in Primary Hyperoxaluria Type 1. [PDF]
DrugsKang C.
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Efficacy and safety of Oxalobacter formigenes in patients with primary hyperoxaluria: A systematic review and meta-analysis of randomized controlled trials. [PDF]
Indian J UrolKhan U +8 more
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Synthesis and hLDHA Inhibitory Activity of New Stiripentol-Related Compounds of Potential Use in Primary Hyperoxaluria. [PDF]
Int J Mol SciRico-Molina M +5 more
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Seminars in Nephrology, 2008
The primary hyperoxalurias (PHs) are rare autosomal-recessive inborn errors of metabolism. In the most severe form (type 1), recurrent kidney stones and progressive nephrocalcinosis lead to the loss of kidney function, accompanied by systemic oxalosis, and often requires dialysis and/or transplantation.
Amy E, Bobrowski, Craig B, Langman
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The primary hyperoxalurias (PHs) are rare autosomal-recessive inborn errors of metabolism. In the most severe form (type 1), recurrent kidney stones and progressive nephrocalcinosis lead to the loss of kidney function, accompanied by systemic oxalosis, and often requires dialysis and/or transplantation.
Amy E, Bobrowski, Craig B, Langman
openaire +2 more sources
Primary Hyperoxaluria in Infancy
Australasian Radiology, 1986ABSTRACTPrimary Hyperoxaluria is a rare autosomal recessive disorder causing progressive renal failure and death before adulthood in most cases1. Acute renal failure due to Primary Hyperoxaluria with renal oxalosis is rare in infancy2‐ 3 and we report such a case emphasizing the importance of ultrasonographic examination in the diagnosis of this ...
H N, Srinivas, C, Ramkumar
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Pediatrics, 1960
The clinical and pathologic findings in a case of primary hyperoxaluria and calcium oxalate nephrocalcinosis in a 7-year-old boy are described and discussed in relation to similar reported cases. The diagnosis was suspected because of nephrocalcinosis in the absence of an abnormality of calcium metabolism and proven by the demonstration of increased ...
Thomas H. Shepard +4 more
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The clinical and pathologic findings in a case of primary hyperoxaluria and calcium oxalate nephrocalcinosis in a 7-year-old boy are described and discussed in relation to similar reported cases. The diagnosis was suspected because of nephrocalcinosis in the absence of an abnormality of calcium metabolism and proven by the demonstration of increased ...
Thomas H. Shepard +4 more
openaire +2 more sources