Results 171 to 180 of about 18,501 (222)
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Journal of the American Veterinary Medical Association, 1981
Summary Pseudorabies was diagnosed in a 5-year-old female crossbred dog by histologic examination and virus isolation. The clinical signs were depression, salivation, head pressing, and emesis. There were no gross pathologic findings. The microscopic findings in the brain stem consisted of mononuclear cell infiltrates in the leptomeninges, subarachnoid
L G, Shell, R W, Ely, R A, Crandell
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Summary Pseudorabies was diagnosed in a 5-year-old female crossbred dog by histologic examination and virus isolation. The clinical signs were depression, salivation, head pressing, and emesis. There were no gross pathologic findings. The microscopic findings in the brain stem consisted of mononuclear cell infiltrates in the leptomeninges, subarachnoid
L G, Shell, R W, Ely, R A, Crandell
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Veterinary Microbiology, 1994
The decrease in titer of PRV antibodies in serum was evaluated at 10, 37, 67, 109 and 173 days of age in 16 non-vaccinated pigs and 43 pigs vaccinated at 3, 67 and 80 days of age with a modified live TK/gIII gene deleted pseudorabies virus (PRV) vaccine.
F, Elvinger +4 more
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The decrease in titer of PRV antibodies in serum was evaluated at 10, 37, 67, 109 and 173 days of age in 16 non-vaccinated pigs and 43 pigs vaccinated at 3, 67 and 80 days of age with a modified live TK/gIII gene deleted pseudorabies virus (PRV) vaccine.
F, Elvinger +4 more
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Specificity of pseudorabies virus serotests
American Journal of Veterinary Research, 1984SUMMARY Pigs experimentally inoculated with bovine herpesvirus-1 or equine herpesvirus-1 developed mild clinical disease signs. Regression of clinical disease was accompanied by development of specific virus-neutralizing antibodies. These antibodies did not react positively with pseudorabies antigens in the serumvirus neutralization test, an indirect ...
J D, Neill, C L, Kelling, M B, Rhodes
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Pseudorabies virus latency: restricted transcription
Archives of Virology, 1990Cloned pseudorabies virus (PRV) sequences representing over 80% of the viral genome were radiolabeled and individually hybridized to nucleic acid in the trigeminal ganglia of acutely and latently infected swine. In acutely infected animals, all cloned probes hybridized to PRV RNA and DNA.
J R, Lokensgard +2 more
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Immunobiology of pseudorabies (Aujeszky's Disease)
Veterinary Immunology and Immunopathology, 1996Aujeszky's Disease (AD), a serious illness of pigs causing significant economic losses in the pig industry, is caused by Pseudorabies Virus (PrV). PrV belongs to the alphaherpesvirus subfamily of the herpesviruses with a double-stranded DNA genome in an enveloped capsid capable of encoding approximately 70 proteins.
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1984
The restriction patterns of genomes of twelve different field isolates of PrV were analyzed. All were found to differ. In most cases, the basis of the differences in restriction patterns was found to lie in the acquisition or deletion of sequences which appear to be nonessential to the productive infection of RK cells. Mutagenesis of a laboratory virus
Tamar Ben-Porat +5 more
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The restriction patterns of genomes of twelve different field isolates of PrV were analyzed. All were found to differ. In most cases, the basis of the differences in restriction patterns was found to lie in the acquisition or deletion of sequences which appear to be nonessential to the productive infection of RK cells. Mutagenesis of a laboratory virus
Tamar Ben-Porat +5 more
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Neurovirulence of pseudorabies virus.
Critical reviews in neurobiology, 1996Virulence is defined as the relative capacity of a microorganism to overcome the defense mechanisms of the host organism and thereby cause disease. Virally induced virulence is usually quantitated by measuring the mean time to death or appearance of symptoms following viral inoculation. In this review we make a distinction between general virulence and
J P, Card, L W, Enquist
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