Results 211 to 220 of about 46,143 (249)

Single-Nucleotide Polymorphisms in Thrombotic Thrombocytopenic Purpura: A Genetic Predisposition to Immune Thrombotic Thrombocytopenic Purpura. [PDF]

Cureus
Konopnicki A, Rischall A, Jana B.
europepmc   +1 more source

Refractory Immune Thrombocytopenic Purpura With Low Immature Platelet Fraction. [PDF]

Cureus
Ali K, Scotto T, Williams JM, Reddy P.
europepmc   +1 more source

A Descriptive 5-Year Analysis of the Demographics and Therapies for Patients With Immune Thrombotic Thrombocytopenic Purpura in the USA: A Multicenter Study of 390 Disease Episodes From 2017 to 2021. [PDF]

J Clin Apher
Jacobs JW, Adkins BD, Adkins BD, Booth GS, Stanek CG, Allen ES, Grossman BJ, Stephens LD, Crowe EP, Daou L, Marques MB, Siniard RC, Wallace LR, Yamada C, Duque MA, Wu Y, Aljuboori O, Harrington TJ, Byrnes DM, Eichbaum Q, Figueroa Villalba CA, Juskewitch JE, Klapper E, Perez-Alvarez I, Klein MS, Aldarweesh F, Alkhateb R, Parsons MG, Schlueter AJ, Tormey CA, Wheeler AP, Powers AA, Webb CB, Yates SG, Bloch EM, Raval JS.   +35 more
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Taylor A, Keogh L, Dickens E, et al. Caplacizumab in pediatric immune thrombotic thrombocytopenic purpura: the UK TTP Registry experience. Blood Adv. 2024;8(17):4563-4567. [PDF]

Blood Adv
europepmc   +1 more source

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Thrombotic microangiopathy, hemolytic uremic syndrome and thrombotic thrombocytopenic purpura: Rare manifestations of Russell’s viper (Daboia russelii) envenoming in Sri Lanka

, 2021
Background: Russell’s viper (Daboia russelii) of Family Viperidae is a highly venomous snake in Sri Lanka and is responsible for the most snakebite deaths. It commonly causes coagulopathy and neuroparalysis. Thrombotic microangiopathy (TMA) including the
R. Rathnayaka, P. Ranathunga, S. Kularatne   +2 more
semanticscholar   +1 more source

Thrombotic Thrombocytopenic Purpura [PDF]

Annual Review of Medicine, 1988
Platelet thrombus formation in small vessels is triggered by certain stimuli, including vascular injury, primary platelet agglutination, or both. The formation and dissolution of platelet thrombi is modulated by proteolysis, plasma factors, PGI2 synthesis and stability, and immune mechanisms.
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Thrombotic Thrombocytopenic Purpura [PDF]

Thrombosis and Haemostasis, 1995
Recent studies indicate that CRTTP patients have excessive shear stress-induced platelet aggregation that is associated with the presence of ULvWF multimers in their plasma and increased vWF-binding to their platelets by flow cytometry. In these CRTTP patients, relapses, excessive shear-aggregation and the presence in their plasma of ULvWF forms are ...
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Thrombotic Thrombocytopenic Purpura

Pediatric Emergency Care, 2011
In 1924, Dr Eli Moschcowitz described a 16-year-old adolescent girl with abrupt onset of petechiae, hemolytic anemia, followed by paralysis, coma, and death. Autopsy showed widespread hyaline thrombi in the terminal arterioles and capillaries of various organs. The syndrome described by Moschowitz is now known as thrombotic thrombocytopenic purpura.
Alex Koyfman, Vincent W. Chiang, Elizabeth Brém   +2 more
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Thrombotic thrombocytopenic purpura

Blood Coagulation & Fibrinolysis, 1992
Thrombotic thrombocytopenic purpura is an uncommon disorder, but it continues to be of considerable interest. The disease mechanisms are unclear and the aetiology is unknown. Perhaps most enigmatic of all, the mode of action of plasma therapy, which successfully induces remission in about two-thirds of cases, is wholly inexplicable. There are currently
Theodore E. Warkentin, William G. Murphy, Jane C. Moore, Catherine P.M. Hayward, John G. Kelton   +4 more
openaire   +3 more sources