Results 111 to 120 of about 1,118,487 (315)

The impact of innate and humoral immune mechanisms on vaccine induced protection against avian influenza H9N2 in broilers

open access: yesScientific Reports
Avian influenza virus (AIV) (H9N2) is a highly contagious avian respiratory disease that causes significant economic losses in the poultry industry sector.
Hesham M. Asaad   +6 more
doaj   +1 more source

Abstracts of the communications presented during the 6th World Rabbit Congress: GENERAL PHYSIOLOGY. Toulouse, July 9-12, 1996

open access: yes, 1996
World Rabbit Science (1996). Abstracts of the communications presented during the 6th World Rabbit Congress: GENERAL PHYSIOLOGY. Toulouse, July 9-12, 1996. World Rabbit Science. 04.
World Rabbit Science
core   +1 more source

Heterozygous loss‐of‐function alleles associate the conserved 3′‐5′ exoribonuclease EXOSC10 with hypersensitivity to the anticancer drug 5‐fluorouracil

open access: yesMolecular Oncology, EarlyView.
EXOSC10, an essential nuclear RNA exosome‐associated 3′‐5′ exoribonuclease, is inhibited by the anticancer drug 5‐fluorouracil (5‐FU), and EXOSC10 depletion increases 5‐FU sensitivity. The colon‐cancer variant EXOSC10S402T, located in a proteolysis motif, is stable and nuclear but nonfunctional in vivo.
Radhika Sain   +10 more
wiley   +1 more source

Abstracts of 2nd International Meeting on Rabbit Biotechnology [PDF]

open access: yes, 2008
World Rabbit Science (2008). Abstracts of 2nd International Meeting on Rabbit Biotechnology. World Rabbit Science. 16(1).
World Rabbit Science   +1 more
core   +1 more source

Cell‐cycle‐specific lesion evolution rather than inhibition of double‐strand‐break repair underpins cisplatin radiosensitization

open access: yesMolecular Oncology, EarlyView.
We analyze cisplatin–DNA adducts (CDAs) and double‐strand breaks (DSBs) in a cell‐cycle‐dependent manner. We find that CDAs form similarly across all cell cycle phases. DSBs arise only in S‐phase. CDAs might not directly impair DSB repair, but S‐phase DSB lesions evolve in the presence of CDAs and disrupt repair in G2, also causing radiosensitization ...
Ye Qiu   +10 more
wiley   +1 more source

Abstracts of the communications presented during the 6th World Rabbit Congress: WOOL AND FUR POIL ET FOURRURE. Toulouse, July 9-12, 1996

open access: yes, 1996
World Rabbit Science (1996). Abstracts of the communications presented during the 6th World Rabbit Congress: WOOL AND FUR POIL ET FOURRURE. Toulouse, July 9-12, 1996. World Rabbit Science. 04.
World Rabbit Science
core   +1 more source

Adaptor protein CIN85 potentiates the motility of osteosarcoma cells via the Akt/mTOR and MMP2‐COL3A1 axis

open access: yesMolecular Oncology, EarlyView.
CIN85 is highly expressed in osteosarcoma, particularly in metastatic lesions. Its overexpression increases cell migration and Matrigel invasion, while silencing CIN85 suppresses these behaviors. Transcriptome analysis shows that CIN85 regulates MMP2, COL3A1, and Akt/mTOR signaling. Targeting these pathways reverses CIN85‐induced motility, highlighting
Iryna Horak   +10 more
wiley   +1 more source

EVALUATION OF EFFICACY COMBINATION OF ZNO NANOPARTICLES AND RESISTED ANTIBIOTICS AGAINST AVIAN PATHOGENIC E.COLI [PDF]

open access: yesAssiut Veterinary Medical Journal
The antibiotic resistance of Avian Pathogenic E. coli is considered one of the biggest public health concerns worldwide and with the growing necessity to find unconventional approaches for formulating new forms of safe and price of effective antibiotics ...
AML BADRY   +5 more
doaj   +1 more source

Abstracts of the Pathology section presented during the "8th World Rabbit Congress". Puebla, Mexico 7-10, 2004

open access: yes, 2005
World Rabbit Science (2006). Abstracts of the Pathology section presented during the "8th World Rabbit Congress". Puebla, Mexico 7-10, 2004. World Rabbit Science. 14(2).
World Rabbit Science
core   +1 more source

Proteasome inhibitor, ixazomib prevents topoisomerase‐I degradation and reverses irinotecan resistance in colorectal cancer

open access: yesMolecular Oncology, EarlyView.
Ixazomib inhibits proteasome‐mediated degradation of topoisomerase I induced by irinotecan, thereby restoring drug sensitivity and promoting tumor cell death in colorectal cancer. Irinotecan, a topoisomerase I (topoI) inhibitor, is widely used for colorectal cancer, but resistance remains a major clinical challenge.
Yuho Ebata   +10 more
wiley   +1 more source

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