Results 61 to 70 of about 8,702 (250)

Debye’s temperature and heat capacity for Sr0.15Ba0.85Bi2Nb2O9 relaxor ferroelectric ceramic [PDF]

open access: yesJournal of Advanced Dielectrics, 2016
A lead-free relaxor ferroelectric, Sr0.15Ba0.85Bi2Nb2O9, was synthesized via solid-state reaction and the temperature-dependence of the heat capacity was measured in a wide temperature range.
A. Peláiz-Barranco   +4 more
doaj   +1 more source

Neutralization of the PTX3/CD44 axis suppresses TGF‐β1 signaling in kidney fibrosis

open access: yesFEBS Letters, EarlyView.
This study investigates how the PTX3/CD44 axis promotes kidney fibrosis by activating TGF‐β1 signaling. We demonstrate that the neutralizing antibody WHC‐001 effectively inhibits this pathway, suppressing myofibroblast activation and partial epithelial–mesenchymal transition (EMT).
Chao‐Chun Cheng   +8 more
wiley   +1 more source

X-ray-induced piezoresponse during X-ray photon correlation spectroscopy of PbMg1/3Nb2/3O3

open access: yesJournal of Synchrotron Radiation
X-ray photon correlation spectroscopy (XPCS) holds strong promise for observing atomic-scale dynamics in materials, both at equilibrium and during non-equilibrium transitions.
Dina Sheyfer   +13 more
doaj   +1 more source

Lead-free relaxor ferroelectrics with 'TTB' structure [PDF]

open access: yes, 2002
Preparation and dielectric characterisations of a great number of TTB-type ceramics have allowed us to show that some of them present relaxor behaviour, due to the occupation of the same crystallographic site by two different cations or anions.
Jean Ravez   +3 more
core   +1 more source

Soft motions encoded by the fold and mediated by conserved hinge sites drive V‐ATPase allosteric machinery and autoinhibition

open access: yesFEBS Letters, EarlyView.
Vacuolar H+‐ATPase (V‐ATPase) is a multisubunit rotary proton pump that acidifies intracellular compartments. Using elastic network models, we identified conserved hinge residues that govern its large‐scale functional motions. We show that intrinsically encoded ‘soft modes’ drive critical functional transitions, including rotary catalysis and ...
Diksha Parwana   +3 more
wiley   +1 more source

Nanoscale ferroelectric/relaxor composites: Origin of large strain in lead-free Bi-based incipient piezoelectric ceramics

open access: yes, 2016
La-doped (Bi0.5Na0.41K0.09)TiO3 ceramics were synthesized using a conventional solid-state reaction route. X-ray diffraction and electric-field-induced polarization studies revealed a phase transformation from a ferroelectric to a relaxor with increasing
Jo, Wook   +12 more
core   +1 more source

Relaxor behavior of (Ba,Sr)(Zr,Ti)O-3 ferroelectric ceramics

open access: yes, 2005
We examine the ferroelectric-relaxor behavior of (Ba0.65Sr0.35)(Zr0.35Ti0.65)O-3 (BSZT) ceramics in the temperature range from 80 to 380 K. A broad dielectric maximum, which shifts to higher temperature with increasing frequency, signifies the relaxor ...
Chan, H.L.W.   +3 more
core   +2 more sources

How structural disorder and polyelectrolyte properties of prothymosin α enable its large interactome

open access: yesFEBS Letters, EarlyView.
In this review, complexes of the fully disordered and highly negatively charged prothymosin α are collected, described, and discussed. Linked to its poly‐electrostatic character, prothymosin α engages a large interactome through multiple different binding modes ranging from fully disordered complexes to short linear motif (SLiM)‐driven interactions ...
Ketty C. Tamburrini   +3 more
wiley   +1 more source

AgNb7O18 : an ergodic relaxor ferroelectric [PDF]

open access: yes, 2014
AgNb7O18 is an ergodic relaxor ferroelectric at room temperature with an incipient transition to the nonergodic state. Electron diffraction confirms a locally polar symmetry, while X-ray diffraction perceives a nonpolar structure.
Richard Beanland   +3 more
core   +1 more source

Mutant p53R273H disrupts PDPK1 homodimerization and contributes to PDPK1 activation

open access: yesMolecular Oncology, EarlyView.
How mutant p53R273H drives AKT signaling is unclear. We show that p53R273H, but not wild‐type, directly binds PDPK1 via a mutation‐dependent conformational change. This interaction disrupts inhibitory PDPK1 homodimerization and enhances AKT phosphorylation.
Mei Chee Lim   +11 more
wiley   +1 more source

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