Results 151 to 160 of about 19,914 (203)
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Journal of Urology, 1984
Most conditions that affect the kidney cause a proportionate and simultaneous loss of glomerular and tubular function. The loss of glomerular function results in the retention of many products of metabolism, including the anions of various organic and inorganic acids, and urea.
T, Pohlman, K A, Hruska, M, Menon
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Most conditions that affect the kidney cause a proportionate and simultaneous loss of glomerular and tubular function. The loss of glomerular function results in the retention of many products of metabolism, including the anions of various organic and inorganic acids, and urea.
T, Pohlman, K A, Hruska, M, Menon
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Pediatric Nephrology, 1990
The term renal tubular acidosis (RTA) is applied to a group of transport defects in the reabsorption of bicarbonate (HCO3-), the excretion of hydrogen ions, or both. On clinical and pathophysiological grounds, RTA can be separated into three main types: distal RTA (type 1), proximal RTA (type 2) and hyperkalaemic RTA (type 4).
J, Rodríguez-Soriano, A, Vallo
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The term renal tubular acidosis (RTA) is applied to a group of transport defects in the reabsorption of bicarbonate (HCO3-), the excretion of hydrogen ions, or both. On clinical and pathophysiological grounds, RTA can be separated into three main types: distal RTA (type 1), proximal RTA (type 2) and hyperkalaemic RTA (type 4).
J, Rodríguez-Soriano, A, Vallo
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Journal of Nephrology, 2006
Abstract: Renal tubular acidosis (RTA) is a form of metabolic acidosis due to abnormal alkali (bicarbonate) loss by the kidneys or their failure to excrete net acid. While the latter does occur in chronic renal failure, the term RTA is usually applied only when the glomerular filtration rate is normal or near normal.
C M, Laing, R J, Unwin
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Abstract: Renal tubular acidosis (RTA) is a form of metabolic acidosis due to abnormal alkali (bicarbonate) loss by the kidneys or their failure to excrete net acid. While the latter does occur in chronic renal failure, the term RTA is usually applied only when the glomerular filtration rate is normal or near normal.
C M, Laing, R J, Unwin
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Medical Clinics of North America, 1983
The kidney has as one of its major responsibilities the excretion of the daily nonvolatile acid load. On a normal American diet, this amounts to about 1 mEq of nonvolatile acid per kilogram body weight. This acid is excreted in the form of titratable acids (e.g., H2PO4 −) and ammonium (NH4 +).
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The kidney has as one of its major responsibilities the excretion of the daily nonvolatile acid load. On a normal American diet, this amounts to about 1 mEq of nonvolatile acid per kilogram body weight. This acid is excreted in the form of titratable acids (e.g., H2PO4 −) and ammonium (NH4 +).
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Annals of Clinical Biochemistry: International Journal of Laboratory Medicine, 1999
Disorders affecting the overall ability of the renal tubules either to secrete hydrogen ions (H+) or to retain bicarbonate ions (HC03) are known collectively by the term renal tubular acidosis (RTA). The condition was first described clinically in 1935,1 confirmed as a renal tubular disorder in 19462 and designated 'renal tubular acidosis' in 1951.3 It
M D, Penney, D A, Oleesky
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Disorders affecting the overall ability of the renal tubules either to secrete hydrogen ions (H+) or to retain bicarbonate ions (HC03) are known collectively by the term renal tubular acidosis (RTA). The condition was first described clinically in 1935,1 confirmed as a renal tubular disorder in 19462 and designated 'renal tubular acidosis' in 1951.3 It
M D, Penney, D A, Oleesky
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Pediatric Clinics of North America, 2019
Renal tubular acidosis should be suspected in poorly thriving young children with hyperchloremic and hypokalemic normal anion gap metabolic acidosis, with/without syndromic features. Further workup is needed to determine the type of renal tubular acidosis and the presumed etiopathogenesis, which informs treatment choices and prognosis.
Robert Todd, Alexander, Martin, Bitzan
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Renal tubular acidosis should be suspected in poorly thriving young children with hyperchloremic and hypokalemic normal anion gap metabolic acidosis, with/without syndromic features. Further workup is needed to determine the type of renal tubular acidosis and the presumed etiopathogenesis, which informs treatment choices and prognosis.
Robert Todd, Alexander, Martin, Bitzan
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Endocrinology and Metabolism Clinics of North America, 1990
Renal tubular acidosis refers to a group of disorders that result from pure tubular damage without concomitant glomerular damage. They could be hereditary (primary) or acquired (secondary to various disease states like sickle cell disease, obstructive uropathy, postrenal transplant, autoimmune disease, or drugs).
M, Rothstein, C, Obialo, K A, Hruska
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Renal tubular acidosis refers to a group of disorders that result from pure tubular damage without concomitant glomerular damage. They could be hereditary (primary) or acquired (secondary to various disease states like sickle cell disease, obstructive uropathy, postrenal transplant, autoimmune disease, or drugs).
M, Rothstein, C, Obialo, K A, Hruska
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Pregnancy and Renal Tubular Acidosis
American Journal of Perinatology, 1999Renal tubular acidosis (RTA) is uncommonly encountered in pregnancy. The risk for these women to develop pregnancy-induced hypertension has not been previously described. The renal defect noted in these women, aggravated by the normal hypervolemia of pregnancy, may predispose to hypertension.
T F, Rowe, K, Magee, F G, Cunningham
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Evaluation of renal tubular acidosis
The Indian Journal of Pediatrics, 2007Renal tubular acidoses (RTA) comprises of a group of disorders characterized by a low capacity for net acid excretion and persistent hyperchloremic, metabolic acidosis. The RTAs are classified into chiefly three types (types 1,2 and 4) based on clinical and laboratory characteristics.
Arvind, Bagga, Aditi, Sinha
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Dimensions of Critical Care Nursing, 2010
Renal tubular acidosis is a relatively uncommon clinical syndrome characterized by the inability of the kidney to adequately excrete hydrogen ions, retain adequate bicarbonate, or both. This syndrome can be categorized into 3 separate disorders, each with unique clinical characteristics.
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Renal tubular acidosis is a relatively uncommon clinical syndrome characterized by the inability of the kidney to adequately excrete hydrogen ions, retain adequate bicarbonate, or both. This syndrome can be categorized into 3 separate disorders, each with unique clinical characteristics.
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