Results 201 to 210 of about 405,671 (296)
Steady-State Concentrations of Glucose-6-Phosphate, 6-Phosphogluconate, and Reduced Nicotinamide Adenine Dinucleotide Phosphate in Strains of Escherichia coli Sensitive and Resistant to Catabolite Repression [PDF]
Abraham W. Hsie+3 more
openalex +1 more source
This study proposes a novel HCC therapy using CA‐4S2@ES‐Cu nanomedicines to amplify cuproptosis, enhancing chemotherapy and immunotherapy. CA‐4 and ES‐Cu synergistically inhibit tumor growth, induce immune responses, and reverse the immunosuppressive microenvironment, offering an effective multidimensional therapeutic strategy for HCC treatment ...
Yingjie Zeng+16 more
wiley +1 more source
Correction: NF-κB regulated expression of A20 controls IKK dependent repression of RIPK1 induced cell death in activated T cells. [PDF]
Layzell S+4 more
europepmc +1 more source
Catabolite repression in Escherichia coli. A study of two hypotheses [PDF]
V. Moses, Michael D. Yudkin
openalex +1 more source
It is reported that Fn14 inhibited EOC metastasis by regulating Slug‐mediated EMT. Furthermore, Fn14 altered the subcellular localization of SIRT2 by interacting with SIRT2, leading to reduced SIRT2 shuttling into the nucleus and subsequently promoting the acetylated degradation of Slug.
Anyue Wu+9 more
wiley +1 more source
Characterization of diverse Cas9 orthologs for genome and epigenome editing. [PDF]
Butterfield GL+9 more
europepmc +1 more source
Catabolite repression of the lac operon. Separate repression of two enzymes [PDF]
Michael D. Yudkin
openalex +1 more source
HDAC6 and USP9X Control Glutamine Metabolism by Stabilizing GS to Promote Glioblastoma Tumorigenesis
Glioblastoma (GBM) growth relies on glutamine synthetase (GS), which is stabilized by histone deacetylase 6 (HDAC6) and deubiquitinated by ubiquitin‐specific peptidase 9, X‐linked (USP9X). HDAC6 promotes GS deacetylation, while USP9X removes its K48‐linked polyubiquitination, enhancing GS stability.
Go Woon Kim+9 more
wiley +1 more source
This work identifies fine‐tuning the expression of PIEZO1 as a critical molecular mechanism underlying the treatment of myocardial infarction by mechanically adapted cardiac patches, which can support the clinical translation of cardiac patch devices.
Yuwen Lu+18 more
wiley +1 more source