Results 71 to 80 of about 67,350 (306)

FSCI 2022

open access: yes, 2023
Course site for FORCE11 Scholarly Communication Institute 2022.
Force11 Scholarly Communication Institute
core   +1 more source

Scholarly Communication in Education Journals [PDF]

open access: yesSocial Science History, 2011
The rise of disciplines is connected with the formation of groups or networks of specialists. It is connected with the emergence of “scientific communities,” theorized about since Thomas Kuhn and Robert Merton. But how is such a community of specialists brought together; how are common orientations among members of a scientific community upheld?
openaire   +2 more sources

Gut Microbiota Regulates Systemic Inflammatory Response and Compensatory Anti‐Inflammatory Response Syndromes by Targeting PF4+ Macrophages in Acute Pancreatitis

open access: yesAdvanced Science, EarlyView.
ABSTRACT Acute pancreatitis (AP) begins with pancreatic local inflammation, leading to the onset of systemic inflammatory response syndrome (SIRS), followed by compensatory anti‐inflammatory response syndrome (CARS), which causes immune paralysis and higher mortality rate.
Liwei Liu   +15 more
wiley   +1 more source

Scholarly publishing and open access: searching for understanding of an emerging is phenomenon

open access: yes, 2007
Scholarly publishing is concerned with the distribution of scholarly information through journals and conferences and other information media. As such scholarly publishing can be understood as a specific part of the information industry.
Kautz, Karlheinz   +2 more
core   +1 more source

GATA4‐Driven Transcription of HtrA1 Promotes Cellular Senescence in Ménière's Disease and Age‐Related Audio‐Vestibular Dysfunction

open access: yesAdvanced Science, EarlyView.
This study identifies the HDAC6/GATA4/HtrA1 axis as a critical driver of cellular senescence in the inner ear. GATA4 nuclear translocation, facilitated by HDAC6 downregulation, transcriptionally activates HtrA1, promoting hair cell senescence, SASP, and audio‐vestibular dysfunction in models of Ménière's disease and age‐related audio‐vestibular ...
Na Zhang   +16 more
wiley   +1 more source

The impact of computer usage on scholarly communication amongst academic social scientists [PDF]

open access: yes, 1999
The study aims to see whether there are differences in the nature and patterns of computer usage for communicating research between disciplines in the social sciences in Brazil and, if so, whether they can be related to factors which can affect the ...
Costa, Sely Maria de Souza
core  

β‐Adrenergic Signaling Promotes Anti‐Tumor Immunity in TP53‐mutant Oral Squamous Cell Carcinoma

open access: yesAdvanced Science, EarlyView.
β‐adrenergic stimulation enhances anti‐tumor immunity in TP53‐deficient oral squamous cell carcinoma by inducing tumor‐derived secretion of CXCL10, which attracts and activates cytotoxic CD8+ T cells. The findings demonstrate that β‐adrenergic signaling alters tumor–immune interactions via CXCL10‐mediated paracrine activation, revealing a neuro‐immune ...
Frederico O. Gleber‐Netto   +20 more
wiley   +1 more source

FSCI 2021

open access: yes, 2023
Course site for Force11 Scholarly Communication Institute 2021. https://www.force11.org/fsci/2021 (Materials stored in the U.S.
Force11 Scholarly Communication Institute
core   +1 more source

Scholarly Communication Initiatives Department Jan 2018 - June 2019 Report

open access: yes, 2019
This report highlights the major accomplishments of the Scholarly Communication Initiatives Department (SCI) with a special emphasis on statistics from Digital Scholarship@UNLV, our institutional repository.
Scholarly Communication Initiatives
core   +1 more source

Tumor‐Derived LAMB3 Drives Immunosuppressive LRRC15+ Fibroblast Formation During Pancreatic Ductal Adenocarcinoma Development

open access: yesAdvanced Science, EarlyView.
A single‐cell atlas of pancreatic ductal adenocarcinoma development reveals progressive ductal‐fibroblast‐immune crosstalk. Tumor‐derived LAMB3 drives the formation of immunosuppressive LRRC15+ fibroblasts through the ITGB1/FAK/MAPK/FOSL2 signaling. Glycolytic reprogramming upregulates LAMB3 and correlates with LRRC15+ fibroblast enrichment.
Xuqing Shi   +23 more
wiley   +1 more source

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