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Intensive receptor blockade and plasma exchange to treat refractory scleroderma renal crisis in patients with agonistic autoantibodies targeting the angiotensin II type 1 and endothelin-1 type A receptors. [PDF]
J Scleroderma Relat DisordHegner B +6 more
europepmc +1 more source
Scleroderma Renal Crisis in a Systemic Sclerosis With Anti-PM/Scl Antibodies. [PDF]
Kidney Int Rep, 2019 Jacquier M +7 more
europepmc +1 more source
Scleroderma renal crisis associated with a complement‑related gene mutation： a case report
Linchuang shenzangbing zazhiDu Qiao-yue, Pan Feng-jun, Wu Wei
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Seminars in Arthritis and Rheumatism, 2015
To discuss the pathophysiology, risk factors, clinical manifestations, diagnosis, treatment, prevention, and outcomes of scleroderma renal crisis (SRC), a serious yet potentially treatable complication of scleroderma (systemic sclerosis).A PubMed search for articles published up until April 2014 was conducted using the following keywords: scleroderma ...
Andres Chiesa-Vottero +1 more
exaly +9 more sources
To discuss the pathophysiology, risk factors, clinical manifestations, diagnosis, treatment, prevention, and outcomes of scleroderma renal crisis (SRC), a serious yet potentially treatable complication of scleroderma (systemic sclerosis).A PubMed search for articles published up until April 2014 was conducted using the following keywords: scleroderma ...
Andres Chiesa-Vottero +1 more
exaly +9 more sources
Scleroderma Renal Crisis Prior to Scleroderma
American Journal of Kidney Diseases, 1989SCLERODERMA renal crisis (SRC) is characterized by rapid onset of severe hypertension and rapid deterioration of renal function. Although this event usually complicates established progressive systemic sclerosis (PSS), occasionally SRC may be the mode of presentation of the illness.
Sharda G Sabnis
exaly +3 more sources
La Presse Médicale, 2021
Scleroderma renal crisis (SRC) is a rare but life-threatening complication of systemic sclerosis (SSc) characterized by malignant hypertension and acute kidney injury. Historically, SRC was the leading cause of death in SSc. However, with the advent of angiotensin converting enzyme (ACE) inhibitors, mortality rates have decreased significantly ...
Marie Hudson +2 more
openaire +2 more sources
Scleroderma renal crisis (SRC) is a rare but life-threatening complication of systemic sclerosis (SSc) characterized by malignant hypertension and acute kidney injury. Historically, SRC was the leading cause of death in SSc. However, with the advent of angiotensin converting enzyme (ACE) inhibitors, mortality rates have decreased significantly ...
Marie Hudson +2 more
openaire +2 more sources
Endothelin-1 expression in scleroderma renal crisis☆
Human Pathology, 2011 The objective of the study was to investigate the role of endothelin-1 in the pathogenesis of scleroderma renal crisis in patients with systemic sclerosis.
Luc Mouthon +2 more
exaly +2 more sources
The Journal of Rheumatology, 2014
Scleroderma renal crisis (SRC) is characterized by malignant hypertension and oligo-anuric acute renal failure. It occurs in 5% of patients with systemic sclerosis (SSc), particularly in patients with diffuse disease during the first years. SRC is more common in patients receiving corticosteroids, the risk increasing with increasing dose.
Luc, Mouthon +4 more
openaire +3 more sources
Scleroderma renal crisis (SRC) is characterized by malignant hypertension and oligo-anuric acute renal failure. It occurs in 5% of patients with systemic sclerosis (SSc), particularly in patients with diffuse disease during the first years. SRC is more common in patients receiving corticosteroids, the risk increasing with increasing dose.
Luc, Mouthon +4 more
openaire +3 more sources
Scleroderma renal crisis: poor outcome despite aggressive antihypertensive treatment
Internal Medicine Journal, 2003 Background: Scleroderma renal crisis (SRC) is a rare but feared complication of scleroderma. Angiotensin-converting enzyme (ACE) inhibition has significantly improved survival, but it is unknown whether prophylactic ACE inhibitors will prevent this ...
J G Walker, M J Ahern, Malcolm D Smith
exaly +2 more sources