Results 61 to 70 of about 44,987 (258)

Open Access: Information crisis … an opportunity for providing information?

open access: yesJournal für Psychologie, 2007
Since the "Berlin Declaration on Open Access to Knowledge in the Sciences and Humanities" was signed by numerous significant scientific organizations in 2003, Open Access – i.e.
Katja Mruck, Günter Mey
doaj  

SSR4 sustains Tertiary Lymphoid Structures by Regulation Quality Control of N‐linked Glycosylation During B‐cell Differentiation Into Plasmacyte in Colorectal Cancer

open access: yesAdvanced Science, EarlyView.
SSR4, a TRAP component induced in B cells, governs BAFFR N‐glycosylation via DDOST to sustain NF‐κB signaling, B‐cell differentiation, and TLS maturation. Its loss impairs anti‐tumor immunity, while overexpression improves antibody glycosylation and ADCC, revealing a critical regulator for cancer immunotherapy.
Wei Zhao   +15 more
wiley   +1 more source

Copyright and self-archiving

open access: yes, 2003
Elizabeth Gadd, Charles Oppenheim, and Steve Probets, The Intellectual Property Rights Issues Facing Self-archiving: Key Findings of the RoMEO Project, <em> D-Lib Magazine </em> , September 2003. Excerpt: &#34;The findings, particularly the Directory of journal publishers&#39; self-archiving policies, should encourage academics ...
openaire   +1 more source

NAT10‐Mediated ac4C Modification of circANKRD12 Reprograms the Tumor Microenvironment

open access: yesAdvanced Science, EarlyView.
NAT10‐dependent acetylation of circANKRD12 drives translation of the circANKRD12_354aa protein, which binds HDAC2 to stabilize c‐Myc via deubiquitination, promoting multiple myeloma (MM) cell proliferation. Concurrently, the circANKRD12‐HDAC2 axis suppresses H3ac‐mediated transcription of IFN‐γ, TNF‐α, and GZMB in NK cells, leading to NK cell ...
Jiale Zhang   +8 more
wiley   +1 more source

Self-archiving on the rise

open access: yes, 2007
Jingfeng Xia and Marija Dalbello, Self-Archiving as an Emergent Scholarly Practice: An Assessment of Self-Archiving in Institutional Repositories, apparently a preprint.  Self-archived May 5, 2007. PS:  Although self-archived in dLIST, only the abstract is free online, at least so far.
openaire   +1 more source

Renal IGFBP6 Interacts With THBS1 to Drive Renal Cellular Senescence and Fibrosis

open access: yesAdvanced Science, EarlyView.
ABSTRACT Epithelial dedifferentiation and myofibroblast activation are critical drivers of chronic kidney disease (CKD) progression. Elevated levels of IGFBP6 have been linked to decreased renal function in CKD patients, but its precise role and underlying mechanisms remain unclear.
Ju‐tao Yu   +26 more
wiley   +1 more source

Harnad on self-archiving

open access: yes, 2003
Stevan Harnad, Self-Archive unto others..., <em> University Affairs </em> , December 2003. A clear and succinct statement of the argument for self-archiving. Excerpt: &#34;Research journal publishers are still stuck in the old system.
openaire   +1 more source

stMixer for Scalable Mosaic Integration and Label Transfer in Spatial Histology and Multi‐Omics

open access: yesAdvanced Science, EarlyView.
stMixer is an unsupervised framework for scalable integration and label transfer across spatial histology and multi‐slide multi‐omics data with incomplete modality overlap. It combines self‐looped cross‐attention, multimodal metric learning, and graph‐guided cluster voting to align heterogeneous sections, correct batch effects, and propagate ...
Qixing Yang   +3 more
wiley   +1 more source

Self-archiving in economics

open access: yes, 2007
Ted Bergstrom and Rosemarie Lavaty, How often do economists self-archive?  A preprint, self-archived February 8, 2007.
openaire   +1 more source

NMI Regulates Adipose Adaptive Thermogenesis Through TLR4/IRF3 Signaling to Promote Obesity

open access: yesAdvanced Science, EarlyView.
Adipose tissue‐derived NMI is secreted under metabolic stress and suppresses adaptive thermogenesis through TLR4/IRF3 signaling, repressing the PPARα/PGC‐1α/UCP1 thermogenic transcriptional program. Genetic ablation or anti ‐ NMI monoclonal antibody treatment enhances energy expenditure, protects against DIO, and ameliorates adipose tissue inflammation,
Ting‐Ting Li   +7 more
wiley   +1 more source

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