Results 121 to 130 of about 29,812 (232)

Phylogenetic analysis of Shiga toxin 1 and Shiga toxin 2 genes associated with disease outbreaks-3 [PDF]

open access: yes, 2011
Copyright information:Taken from "Phylogenetic analysis of Shiga toxin 1 and Shiga toxin 2 genes associated with disease outbreaks"http://www.biomedcentral.com/1471-2180/7/109BMC Microbiology 2007;7():109-109.Published online 4 Dec 2007PMCID:PMC2211750.
Larry D Farrell (66913)   +5 more
core   +1 more source

Genomic analyses reveal two distinct lineages of Corynebacterium ulcerans strains

open access: yesNew Microbes and New Infections, 2018
Corynebacterium ulcerans is an important zoonotic pathogen which is causing diphtheria-like disease in humans globally. In this study, the genomes of three recently isolated C.
R. Subedi   +8 more
doaj   +1 more source

Shiga Toxin-Associated Hemolytic Uremic Syndrome and Unexpected Course

open access: yesTurkish Journal of Nephrology, 2019
Hemolytic uremic syndrome is a disease that often requires the transfusion of blood products. A child (two and a half years old, boy) with Shiga toxin-associated hemolytic uremic syndrome who developed respiratory distress after transfusion is presented.
Hakan ERDOĞAN   +4 more
doaj  

Immunomodulation by shiga toxin 2 [PDF]

open access: yes, 2010
The Shiga-like toxins have DNA sequence homology to the toxins accountable for the dysentery brought about by the Shigella species. Escherichia coli which encode and produce shiga-like toxins are referred to as shiga toxin-producing E.
Chu, Audrey
core  

Microvesicle Involvement in Shiga Toxin-Associated Infection [PDF]

open access: yes, 2017
Shiga toxin is the main virulence factor of enterohemorrhagic Escherichia coli, a non-invasive pathogen that releases virulence factors in the intestine, causing hemorrhagic colitis and, in severe cases, hemolytic uremic syndrome (HUS).
Tontanahal, Ashmita   +5 more
core   +1 more source

Exploiting the retrograde transport of disarmed toxins for the delivery of exogenous antigens into MHC class 1 presentation pathway [PDF]

open access: yes
The targeting of exogenous antigen into the MHC class 1-restricted presentation pathway is required for the induction of cytotoxic T lymphocytes (CTL) which have been shown to be an important component of the protective response to intracellular ...
Noakes, Karen
core  

Characterization of non -Shiga toxin- producing Escherichia coli O157 strains isolated from dogs Caracterización de cepas de Escherichia coli O157 no productoras de toxina Shiga aisladas de perros

open access: yesRevista Argentina de Microbiología, 2010
Shiga toxin-negative Escherichia coli O157 strains of various H types have been associated with diarrhea in children and are considered potentially pathogenic for humans. In this study, we describe non-Shiga toxin-producing E. coli O157 E.
A. Bentancor   +7 more
doaj  

Escherichia coli O157:H7 Pembawa Shiga Like Toxin [PDF]

open access: yes, 2014
Abstrak: Eschericha coli O157:H7 merupakan strain Enterohemorrhagic Escherichia coli (EHEC), patogen yang dapat menyebabkan kolitis hemoragik, sindrom uremia hemolitik, penyakit gagal ginjal akut disertai dengan anemia hemolotik mikroangiopatik dan ...
Pasaribu, Donna Mesina Rosadini
core  

In Vivo Formation of Hybrid Toxins Comprising Shiga Toxin and the Shiga-Like Toxins and Role of the B Subunit in Localization and Cytotoxic Activity [PDF]

open access: yes, 1989
Shiga toxin, Shiga-like toxin I (SLT-I) and Shiga-like toxin II (SLT-H) are cell-associated cytotoxins that kill both Vero cells and HeLa cells, whereas Shiga-like toxin II variant (SLT-llv) is an extracellular cytotoxin that is more cytotoxic for Vero ...
O\u27Brien, Alison D.   +4 more
core   +1 more source

Expression of cholera toxin B subunit in Saccharomyces cerevisiae [PDF]

open access: yes
Cholera toxin, secreted by Vibrio cholerae, consists of A and B subunits. Cholera toxin B subunit (CTB) is used in many scientific research. It has already been expressed in several bacterial and plant systems.
ارزنلو, محسن   +6 more
core  

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