Results 111 to 120 of about 41,221 (298)
Nonnegative signed total Roman domination in graphs
Let $G$ be a finite and simple graph with vertex set $V(G)$. A nonnegative signed total Roman dominating function (NNSTRDF) on a graph $G$ is a function $f:V(G)\rightarrow\{-1, 1, 2\}$ satisfying the conditions that (i) $\sum_{x\in N(v)}f(x)\ge 0$ for
Nasrin Dehgardi, Lutz Volkmann
doaj +1 more source
Data in the social sciences can often modeled using signed graphs, graphs where every edge has a sign + or −, or marked graphs, graphs where every vertex has a sign + or −.
Kota Reddy, M S Subramanya, P Siva
core
Signed circuit $6$-covers of signed $K_4$-minor-free graphs
Bermond, Jackson and Jaeger [{\em J. Combin. Theory Ser. B} 35 (1983): 297-308] proved that every bridgeless ordinary graph $G$ has a circuit $4$-cover and Fan [{\em J. Combin. Theory Ser.
Zhang, Cun-Quan +3 more
core
Diagonal Forms, Linear Algebraic Methods and Ramsey-Type Problems [PDF]
This thesis focuses mainly on linear algebraic aspects of combinatorics. Let N_t(H) be an incidence matrix with edges versus all subhypergraphs of a complete hypergraph that are isomorphic to H. Richard M.
Wong, Wing Hong Tony
core +1 more source
The proliferation of signed networks in contemporary social media platforms necessitates robust privacy-preserving mechanisms. Graph unlearning, which aims to eliminate the influence of specific data points from trained models without full retraining, becomes particularly critical in these scenarios where user interactions are sensitive and dynamic ...
Zhifei Luo +3 more
openaire +2 more sources
Loss of IGF‐1R impairs DNA‐PKcs recruitment to chromatin leading to defective end‐joining
IGF‐1R promotes radioresistance by facilitating DNA‐PKcs recruitment to chromatin, enabling non‐homologous end‐joining (NHEJ) repair of double‐strand breaks. Inhibition or loss of IGF‐1R disrupts this recruitment to damage sites, driving compensatory reliance on microhomology‐mediated end‐joining (MMEJ) repair.
Matthew O. Ellis +3 more
wiley +1 more source
MITF maintains genome stability in nonmelanocyte lineages
MITF is essential for melanocyte survival and acts as an oncogene in 10%–20% of melanomas. We show that MITF depletion causes genome instability in nonmelanocytic cells, leading to LATS2‐mediated P53 activation, cell cycle arrest, and apoptosis. This study highlights the role of MITF as a genome maintenance factor beyond the melanocyte lineage. Created
Drifa H. Gudmundsdottir +13 more
wiley +1 more source
Oncogenic DMTF1β promotes cancer cell motility by regulating autophagy through ULK1 stabilization
In the current study, we demonstrate that the oncogene DMTF1β regulates ULK1 stability by reducing its proteasomal degradation in cancer cells. This stabilization enables ULK1 to induce autophagy, which in turn facilitates cancer cell migration. Consequently, reduced DMTF1β levels lead to decreased autophagy and impaired cancer cell migration.
Jun Xu +13 more
wiley +1 more source
In this paper we introduced a new notion radial signed graph of a signed graph and its properties are obtained. Also, we obtained the structural characterization of radial signed graphs.
P.Siva Kota Reddy
core +1 more source

