Results 201 to 210 of about 965,762 (355)
A Skin‐Adherent Magneto‐Inertial Wearable for Real‐Time Joint Motion Analysis
An imperceptible magneto‐inertial wearable couples a skin‐adherent PDMS‐NdFeB patch with a 9‐DoF IMU to track joint kinematics in real time and classify range‐of‐motion (Limited/Normal/Hypermobile) via a 1D‐CNN (95.3% accuracy). Wireless BLE streaming and a mobile app enable feedback.
Montserrat Ramirez‐De Angel +2 more
wiley +1 more source
These findings suggest that UCP2 promotes LD formation and release in acinar cells by upregulating ACSL3 expression. This alteration in the local lipid metabolic environment indirectly drives the MMT process. Additionally, UCP2 may regulate the acetylation of Smad3 through Sirt1, enhancing its nuclear activity and activating the TGF‐β/ Smad3 signaling ...
Kunpeng Wang +9 more
wiley +1 more source
A Comparison of Skeletal Muscle Diffusion Tensor Imaging Tractography Seeding Methods. [PDF]
Damon BM +3 more
europepmc +1 more source
Further observations upon the release of histamine by skeletal muscles
G. V. Anrep +3 more
openalex +2 more sources
Ma et al. report the hippocampal and hepatic acetylcholine (ACh) levels are differently modulated by regular versus irregular exercise, which contributes to coordinate hippocampal astrocyte activity and hepatic FBXL6high neutrophil recruitment. This ACh‐modulated brain‐liver circuit links the effects of exercise on fear memory extinction and liver ...
Pengjiao Ma +10 more
wiley +1 more source
Overlaps of Skeletal Muscle Fatigue and Skeletal Muscle Damage: The Muscle Injury Continuum. [PDF]
Schwiete C +4 more
europepmc +1 more source
PRDM16 Reduces Cellular Senescence by Upregulating GSTM1
Cellular senescence drives aging and aging‐related organ disorders, yet PRDM16's role remains unexplored. This work uncovers that PRDM16 decreases significantly in aged organs, while its loss accelerates cellular senescence and aging‐related organ injury.
Qian Yuan +7 more
wiley +1 more source
Signaling networks governing skeletal muscle growth, atrophy, and cachexia : By. [PDF]
Joshi AS +3 more
europepmc +1 more source

