Results 101 to 110 of about 15,576,902 (304)

Boston University School of Medicine catalog [PDF]

open access: yes, 1896
Course catalog and informational material for the Boston University School of ...
Boston University School of Medicine
core   +1 more source

Boston University School of Medicine catalog [PDF]

open access: yes, 1875
Course catalog and informational material for the Boston University School of ...
Boston University School of Medicine
core  

Microbiome‐blood–brain barrier interactions in aging — mechanisms and therapeutic potential

open access: yesFEBS Letters, EarlyView.
Aging reshapes the gut microbiome (↓SCFA‐producing commensals; ↑pro‐inflammatory outputs), shifting circulating metabolites (↓SCFAs; ↑LPS, ↑TMAO, ↑PAA) that act at the BBB to increase nonspecific transcytosis, alter transport, and promote astrocyte reactivity, heightening brain vulnerability.
Daniel Cuervo‐Zanatta   +3 more
wiley   +1 more source

THE SOCIAL TREND IN MEDICINE [PDF]

open access: yesJournal of the American Medical Association, 1920
ABSTRACT Before the war, a new era made itself known in many ways; but medicine, especially in the United States, either held aloof or, at any rate, did not actively face the facts. Many things included under the term socialization affected medical practice as well as medical study.
openaire   +1 more source

XII Congreso Latinoamericano de Medicina Social y Salud Colectiva [PDF]

open access: yes, 2012
Association of Social Medicine, Latin American
core   +1 more source

Boston University School of Medicine catalog [PDF]

open access: yes, 1893
Course catalog and informational material for the Boston University School of ...
Boston University School of Medicine
core  

An epithelial GPR35 isoform supports tumor‐associated transcriptional and metabolic phenotypes

open access: yesFEBS Letters, EarlyView.
GPR35 generates two functionally distinct isoforms with previously unresolved roles. GPR35‐short mediates immune‐cell chemotaxis, while GPR35‐long is enriched in colorectal cancer epithelium, where it supports increased metabolism, proliferation, and tumor‐associated transcriptional programs.
Jørgen D. Rønneberg   +14 more
wiley   +1 more source

Boston University School of Medicine catalog [PDF]

open access: yes, 1899
Course catalog and informational material for the Boston University School of ...
Boston University School of Medicine
core  

Nutrient/TOR signaling controls adipose mitochondrial transcription factor A (TFAM) to regulate organismal growth in Drosophila

open access: yesFEBS Letters, EarlyView.
Animals must match their growth rate to available nutrients. We show that in Drosophila larvae, the nutrient‐sensing TOR kinase controls growth by regulating levels of TFAM, a key regulator of mitochondrial function, in the adipose tissue. When nutrients are abundant, high TOR activity suppresses TFAM, lowering mitochondrial bioenergetic activity and ...
Shrivani Sriskanthadevan‐Pirahas   +4 more
wiley   +1 more source

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