Results 221 to 230 of about 164,048 (382)

Neurophysiological Effect of Transcutaneous Electrical Spinal Cord Stimulation in Chronic Complete Spinal Cord Injury

open access: yesArtificial Organs, EarlyView.
TESCS‐ABT induced neurophysiological changes in individuals with chronic complete SCI, however these did not consistently translate into functional improvements. These findings highlight the role of residual supraspinal connectivity and inform future research to optimise this neurotechnology for SCI rehabilitation and identify likely responders ...
E. L. McNicol   +6 more
wiley   +1 more source

SCIENCE AND SPASTICITY [PDF]

open access: yesDevelopmental Medicine & Child Neurology, 1993
openaire   +2 more sources

Slower Pace of Intellectual Development Is Common in Children With Cerebral Palsy–A Population‐Based Study

open access: yesActa Paediatrica, EarlyView.
ABSTRACT Aim To describe the intellectual level and changes during development in children with cerebral palsy (CP) and to investigate if there are factors associated with the pace of intellectual development. Method Population‐based study of all 264 children (141 boys, 123 girls) with CP born 1999–2006 in the region of Västra Götaland. Information was
Mattias Wicke Selvén   +2 more
wiley   +1 more source

The return of metabolism: biochemistry and physiology of glycolysis

open access: yesBiological Reviews, EarlyView.
ABSTRACT Glycolysis is a fundamental metabolic pathway central to the bioenergetics and physiology of virtually all living organisms. In this comprehensive review, we explore the intricate biochemical principles and evolutionary origins of glycolytic pathways, from the classical Embden–Meyerhof–Parnas (EMP) pathway in humans to various prokaryotic and ...
Nana‐Maria Grüning   +19 more
wiley   +1 more source

From Genomic and Epigenomic Maps to Medicines in Adult T‐Cell Leukemia/Lymphoma

open access: yesCancer Science, EarlyView.
ABSTRACT Adult T‐cell leukemia/lymphoma (ATL) is an aggressive and refractory hematologic malignancy that is caused by human T‐cell leukemia virus type‐1 (HTLV‐1) retrovirus. ATL results from a combination of viral latency and the accumulation of abnormalities throughout the genome, epigenome, transcriptome, and signaling pathways.
Kako Suzuki, Makoto Yamagishi
wiley   +1 more source

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