Results 121 to 130 of about 64,475 (321)

AGES OF 70 DWARFS OF THREE POPULATIONS IN THE SOLAR NEIGHBORHOOD: CONSIDERING O AND C ABUNDANCES IN STELLAR MODELS [PDF]

, 2016
Zhishuai Ge, S. L. Bi, Yuquan Chen, T. D. Li, Jingkun Zhao, K. Liu, Jason W. Ferguson, Yaqian Wu   +7 more
openalex   +1 more source

Survivin and Aurora Kinase A control cell fate decisions during mitosis

Molecular Oncology, EarlyView.
Aurora A interacts with survivin during mitosis and regulates its centromeric role. Loss of Aurora A activity mislocalises survivin, the CPC and BubR1, leading to disruption of the spindle checkpoint and triggering premature mitotic exit, which we refer to as ‘mitotic slippage’.
Hana Abdelkabir, Shalitha Wickrama Arachchige, Sally P. Wheatley   +2 more
wiley   +1 more source

The Sun Remains Relatively Refractory Depleted: Elemental Abundances for 17,412 Gaia RVS Solar Analogs and 50 Planet Hosts

The Astrophysical Journal
The element abundances of stars, particularly the refractory elements (e.g., Fe, Si, and Mg), play an important role in connecting stars to their planets. Most Sun-like stars do not have refractory abundance measurements since obtaining a large sample of
Rayna Rampalli, Melissa K. Ness, Graham H. Edwards, Elisabeth R. Newton, Megan Bedell   +4 more
doaj   +1 more source

Observable Effects of Rotation on Stellar Abundances [PDF]

, 1993
S. Balachandran
openalex   +1 more source

CDK11 inhibition induces cytoplasmic p21WAF1 splice variant by p53 stabilisation and SF3B1 inactivation

Molecular Oncology, EarlyView.
CDK11 inhibition stabilises the tumour suppressor p53 and triggers the production of an alternative p21WAF1 splice variant p21L, through the inactivation of the spliceosomal protein SF3B1. Unlike the canonical p21WAF1 protein, p21L is localised in the cytoplasm and has reduced cell cycle‐blocking activity.
Radovan Krejcir, Lukasz Arcimowicz, Lucia Martinkova, Vaclav Hrabal, Filip Zavadil Kokas, Tomas Henek, Martina Kucerikova, Ondrej Bonczek, Pavlina Zatloukalova, Lenka Hernychova, Philip J. Coates, Borivoj Vojtesek, David P. Lane   +12 more
wiley   +1 more source

Stellar Abundances in Dwarf Irregular Galaxies

, 2003
Dwarf irregular galaxies appear to have undergone very slow chemical evolution since they have low nebular abundances, but have had ongoing star formation over the past 15 Gyr. They are too distant for red giant abundance analyses to examine the details of their chemical evolution, however the isolated, bright blue supergiants do allow us to determine ...
Venn, K.A., Tolstoy, E., Kaufer, A., Kudritzki, R.P.   +3 more
openaire   +3 more sources

Reduced vascular leakage correlates with breast carcinoma T regulatory cell infiltration but not with metastatic propensity

Molecular Oncology, EarlyView.
A mouse model for vascular normalization and a human breast cancer cohort were studied to understand the relationship between vascular leakage and tumor immune suppression. For this, endothelial and immune cell RNAseq, staining for vascular function, and immune cell profiling were employed.
Liqun He, Chiara Testini, Neda Hekmati, Altea Bonello, Aglaia Schiza, Emmanuel Nwadozi, Mia Phillipson, Carina Strell, Michael Welsh   +8 more
wiley   +1 more source

Observations of the Ultraviolet-bright Star III-60 in the Globular Cluster NGC 6723

The Astrophysical Journal
We have analyzed archival far-ultraviolet spectra of the UV-bright star III-60 in the globular cluster NGC 6723 obtained with the Far Ultraviolet Spectroscopic Explorer and the Cosmic Origins Spectrograph. We find that the star's photospheric parameters (
William V. Dixon
doaj   +1 more source

Spectroscopic Determination of Stellar Parameters and Oxygen Abundances for Hyades/Field G–K Dwarfs [PDF]

, 2020
Yoichi Takeda, Satoshi Honda
openalex   +1 more source

Class IIa HDACs forced degradation allows resensitization of oxaliplatin‐resistant FBXW7‐mutated colorectal cancer

Molecular Oncology, EarlyView.
HDAC4 is degraded by the E3 ligase FBXW7. In colorectal cancer, FBXW7 mutations prevent HDAC4 degradation, leading to oxaliplatin resistance. Forced degradation of HDAC4 using a PROTAC compound restores drug sensitivity by resetting the super‐enhancer landscape, reprogramming the epigenetic state of FBXW7‐mutated cells to resemble oxaliplatin ...
Vanessa Tolotto, Nicolò Gualandi, Ylenia Cortolezzis, Raffaella Picco, Monica Colitti, Francesca D'Este, Mariachiara Gani, Wayne W. Hancock, Giovanni Terrosu, Cristina Degrassi, Francesca Agostini, Claudio Brancolini, Luigi E. Xodo, Eros Di Giorgio   +13 more
wiley   +1 more source