Results 81 to 90 of about 8,529,264 (233)

p190A/ARHGAP35 and p190B/ARHGAP5 proteins in endometrial cancer: a novel cancer‐relevant paralog interplay

open access: yesMolecular Oncology, EarlyView.
This study identifies ARHGAP5, in addition to the frequently mutated ARHGAP35, as significantly mutated in endometrial cancer. Mutations in both genes co‐occur and are associated with their correlated downregulation. Functional CRISPR studies show that both paralogs regulate similar pathways, including actin cytoskeleton organization.
Mathilde Pinault   +12 more
wiley   +1 more source

A Note on |A|k Summability Factors for Infinite Series

open access: yesJournal of Inequalities and Applications, 2007
We obtain sufficient conditions on a nonnegative lower triangular matrix A and a sequence λn for the series ∑anλn/nann to be absolutely summable of order k≥1 by A.
Ekrem Savaş, B. E. Rhoades
doaj   +1 more source

Extracellular matrix remodeling and immune reprogramming drive residual tumor progression of liver cancer after incomplete microwave ablation

open access: yesMolecular Oncology, EarlyView.
Incomplete microwave ablation (iMWA) of liver cancer triggers a biphasic progression in residual tumors. At Day 3, the microenvironment is characterized by acute inflammatory responses and extracellular matrix (ECM) remodeling. By Day 14, a profound shift occurs toward oncogenic signal transduction and immunosuppression, marked by macrophage ...
Yu Liu   +9 more
wiley   +1 more source

Characterization of Absolute Summability Factors

open access: yesJournal of Mathematical Analysis and Applications, 1995
zbMATH Open Web Interface contents unavailable due to conflicting licenses.
Sarıgöl, Mehmet Ali, Bor, Hüseyin
openaire   +3 more sources

Mutant p53R273H disrupts PDPK1 homodimerization and contributes to PDPK1 activation

open access: yesMolecular Oncology, EarlyView.
How mutant p53R273H drives AKT signaling is unclear. We show that p53R273H, but not wild‐type, directly binds PDPK1 via a mutation‐dependent conformational change. This interaction disrupts inhibitory PDPK1 homodimerization and enhances AKT phosphorylation.
Mei Chee Lim   +11 more
wiley   +1 more source

Absolute matrix summability via almost increasing sequence

open access: yes, 2020
In the present paper, a new theorem concerning absolute matrix summability factors, which generalizes a theorem on the summability factors of infinite series, has been proved by using almost increasing sequence and matrix ...
Kartal, Bağdagül, Özarslan, Hikmet
core   +1 more source

On the absolute Nörlund summability factors [PDF]

open access: yes, 2000
In this paper a theorem on the absolute Nörlund summability factors has been proved under more weaker conditions by using an almost increasing ...
Bor, H.
core  

The impact of leukocyte‐associated RNA retention on platelet RNA‐based cancer detection

open access: yesMolecular Oncology, EarlyView.
Platelets carry RNA signals that may help detect cancer, but platelet samples can also retain RNA from white blood cells. We found that this retained white blood cell signal did not significantly affect machine learning cancer classification, suggesting that the cancer‐related RNA signal in platelets is largely independent of leukocyte‐associated RNA ...
Michał Sieczczyński   +12 more
wiley   +1 more source

On the Summability Factors of Infinite Series

open access: yesCommunications, Faculty Of Science, University of Ankara Series A1Mathematics and Statistics, 1983
The author establishes the following theorem on the summability factors of infinite series. If (i) \(\lambda_ n=o(1)\), (ii) \(\sum^{n}_{v=1}v \log v| \Delta^ 2\lambda_ v| =O(1)\), \(n\to \infty\) and if there exists a \(\beta >0\) such that the sequence \((n^{\beta -k}| \phi_ n|^ k)\) is non-increasing and \(\sum^{n}_{v=1}v^{- k}| \phi_ vt_ v|^ k=O ...
openaire   +2 more sources

Overcoming platinum resistance by the disulfiram metabolite CuET in novel preclinical models of human carcinomas

open access: yesMolecular Oncology, EarlyView.
Cisplatin‐resistant cancer cells survive DNA‐damaging platinum‐based treatment yet remain sensitive to CuET‐induced proteotoxic stress. The distinct mechanism of action allows CuET to overcome and eliminate cisplatin‐resistant tumour cells, unless highly expressed metallothioneins (MTs) interfere with CuET, thereby highlighting MT levels as candidate ...
Stefanos Arampatzis   +20 more
wiley   +1 more source

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