Results 151 to 160 of about 21,364,816 (201)

Author Correction: Synaptotagmin-7 outperforms synaptotagmin-1 to promote the formation of large, stable fusion pores via robust membrane penetration. [PDF]

Nat Commun
Courtney KC, Mandal T, Mehta N, Wu L, Li Y, Das D, Cui Q, Chapman ER.   +7 more
europepmc   +1 more source

Cellular mechanisms of long-term osmoregulation in magnocellular neurons. [PDF]

J Neuroendocrinol
Haan KD, Fisher TE.
europepmc   +1 more source

Gut-brain axis: beneficial impact of Shouchella clausii spores on fructose induced dysfunction is associated with modulation of the deoxycholic acid - TGR5 pathway. [PDF]

Mol Med
Spagnuolo MS, Petecca N, De Palma F, Troise AD, Di Porzio A, Barrella V, Saggese A, De Pascale S, De Stefano M, Scaloni A, Baccigalupi L, Ricca E, Iossa S, Mazzoli A, Cigliano L.   +14 more
europepmc   +1 more source

Hippocampal small RNAs from patients with schizophrenia induce specific cognitive and neural phenotypes in mice. [PDF]

Cell Death Discov
Galán-Ganga M, Guisado-Corcoll A, Sancho-Balsells A, Herrero-Lorenzo M, Ballasch I, Patterer L, Alberch J, Arranz B, Giralt A, Martí E.   +9 more
europepmc   +1 more source

Restricting Synaptotagmin-3 Internalization Mitigates Cerebral Ischemia/Reperfusion Injury by Curtailed Neuronal Apoptosis and Microglial Re-Programming. [PDF]

CNS Neurosci Ther
Xu H, Mi H, Cheng R, Gui T, Hu F, Yang Y, Cheng J, Xue Q.   +7 more
europepmc   +1 more source

tDCS improves early Alzheimer's disease by synaptic vesicle fusion and release. [PDF]

Mil Med Res
Zhuang YY, Yan JM, Wu TC, Xu WS, Wu B, Xie X, Wang WJ, Lin HW, Jian JW, Wang JZ, Jiang T, Chen LM, Qiu YX, Hu ZY, Zhou YH, Yang T, Yang MG, Zhu JF, Tao J, Chen LD, Li WG, Yan K, Liu WL.   +22 more
europepmc   +1 more source

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Synaptotagmin I is a molecular target for lead

Journal of Neurochemistry, 2001
Lead poisoning can cause a wide range of symptoms with particularly severe clinical effects on the CNS. Lead can increase spontaneous neurotransmitter release but decrease evoked neurotransmitter release. These effects may be caused by an interaction of lead with specific molecular targets involved in neurotransmitter release.
C M, Bouton, L P, Frelin, C E, Forde, H, Arnold Godwin, J, Pevsner   +4 more
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Synaptotagmin I and 1B4 are identical: implications for Synaptotagmin distribution in the primate brain

Neuroscience Letters, 1995
We have determined that the human cDNA sequence of the previously described primate brain mRNA species 1B4 is nearly identical (99.95% similarity) to that of human Synaptotagmin I. The apparent identity of Synaptotagmin I with 1B4, whose distribution in the brain of the monkey Cynomolgous was determined previously by Northern blot and in situ ...
D, Chowdhury, G H, Travis, J G, Sutcliffe, F H, Burton   +3 more
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Calcium‐Dependent Self‐Association of Synaptotagmin I

Journal of Neurochemistry, 1996
Abstract: Synaptotagmin I, an integral membrane protein of secretory vesicles, appears to have an essential role in calcium‐triggered hormone and neurotransmitter release. The large cytoplasmic domain of synaptotagmin I has two C2 domains that are thought to mediate calcium and phospholipid binding.
C K, Damer, C E, Creutz
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