Results 231 to 240 of about 8,006,579 (361)

Separation of blast cell and T-lymphocyte progenitors in the blood of patients with acute myeloblastic leukemia [PDF]

, 1979
MD Minden, RN Buick, E. A. McCulloch
openalex   +1 more source

Integration of single‐cell and bulk RNA‐sequencing data reveals the prognostic potential of epithelial gene markers for prostate cancer

Molecular Oncology, EarlyView.
Prostate cancer is a leading malignancy with significant clinical heterogeneity in men. An 11‐gene signature derived from dysregulated epithelial cell markers effectively predicted biochemical recurrence‐free survival in patients who underwent radical surgery or radiotherapy.
Zhuofan Mou, Lorna W. Harries
wiley   +1 more source

The Structural Biology of T-Cell Antigen Detection at Close Contacts. [PDF]

Immunol Rev
Lui Y, Ferreira Fernandes J, Vuong MT, Sharma S, Santos AM, Davis SJ.   +5 more
europepmc   +1 more source

Clinical significance of stratifying prostate cancer patients through specific circulating genes

Molecular Oncology, EarlyView.
We tested a specific panel of genes representative of luminal, neuroendocrine and stem‐like cells in the blood of prostate cancer patients, showing predictive value from diagnosis to late stages of disease. This approach allows monitoring of treatment responses and outcomes at specific time points in trajectories.
Seta Derderian, Edouard Jarry, Arynne Santos, Quentin Vesval, Lucie Hamel, Rafael Sanchez‐Salas, Alexis Rompré‐Brodeur, Wassim Kassouf, Raghu Rajan, Fadi Brimo, Marie Duclos, Armen Aprikian, Simone Chevalier   +12 more
wiley   +1 more source

Modern Approach to Nodal T-Cell Lymphomas. [PDF]

Adv Anat Pathol
Ondrejka SL, de Leval L, de Leval L.
europepmc   +1 more source

MET variants with activating N‐lobe mutations identified in hereditary papillary renal cell carcinomas still require ligand stimulation

Molecular Oncology, EarlyView.
MET variants in the N‐lobe of the kinase domain, found in hereditary papillary renal cell carcinoma, require ligand stimulation to promote cell transformation, in contrast to other RTK variants. This suggests that HGF expression in the microenvironment is important for tumor growth in such patients. Their sensitivity to MET inhibitors opens the way for
Célia Guérin, Audrey Vinchent, Marie Fernandes, Isabelle Damour, Agathe Laratte, Rémi Tellier, Gabriella O. Estevam, Jean‐Pascal Meneboo, Céline Villenet, Clotilde Descarpentries, James S. Fraser, Martin Figeac, Alexis B. Cortot, Etienne Rouleau, David Tulasne   +14 more
wiley   +1 more source

Cell cycle, cell division, cell death

Molecular Biology of the Cell, 2019
Jan M. Skotheim, Amy Shaub Maddox
openaire   +2 more sources

Impact of genotypic variability of measles virus T-cell epitopes on vaccine-induced T-cell immunity. [PDF]

NPJ Vaccines
Emmelot ME, Bodewes R, Maissan C, Vos M, de Swart RL, van Els CACM, Kaaijk P.   +6 more
europepmc   +1 more source

NUCLEOSIDE-PHOSPHORYLASE DEFICIENCY, AUTOIMMUNE HAEMOLYTIC ANAE MIA AND SELECTIVE T-CELL DEFICIENCY [PDF]

, 1978
E. Carapella‐de Luca, F Aiuti, P. Lucarelli, Monica Tozzi, P Vignetti, L. Bruni, Dirk Roos, Rosa Maria Corbo, C Imperato   +8 more
openalex   +1 more source

Etoposide‐induced cancer cell death: roles of mitochondrial VDAC1 and calpain, and resistance mechanisms

Molecular Oncology, EarlyView.
The complex mode of action of the topoisomerase II inhibitor etoposide in triggering apoptosis involves several mechanisms: overexpression of the mitochondrial protein VDAC1, leading to its oligomerization and formation of a large channel that mediates the release of pro‐apoptotic protein; and overexpression of the apoptosis regulators p53, Bax, and ...
Aditya Karunanithi Nivedita, Varda Shoshan‐Barmatz   +1 more
wiley   +1 more source