Analysis of neuronal cardiolipin and monolysocardiolipin from biological samples with cyclic ion mobility mass spectrometry. [PDF]
Front PhysiolEmaus KJ +5 more
europepmc +1 more source
3D bioprinted myocardium patches for rare Barth syndrome: TAZ mutation correction in cardioblasts. [PDF]
Ann Med Surg (Lond)Tahir A, Imtiaz E, Mahato RK.
europepmc +1 more source
Plasmalogens as biomarkers and therapeutic targets. [PDF]
J Lipid ResCurran CS, Remaley AT, Torabi-Parizi P.
europepmc +1 more source
Mitochondrial membrane lipids in the regulation of bioenergetic flux. [PDF]
Cell MetabDecker ST, Funai K.
europepmc +1 more source
Mitochondrial cardiolipin remodeling facilitates efficient myoblast differentiation. [PDF]
J Lipid ResOhba Y, Fujiwara CA, Arita M.
europepmc +1 more source
Mitochondrial cristae remodeling: Mechanisms, functions, and pathology. [PDF]
Cell InsightYu J +7 more
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Beyond Powerhouses: Roles of Mitochondria, from Development to Therapeutic Potential. [PDF]
Biology (Basel)Pereira SP, Tavares LC.
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Contemporary insights into elamipretide's mitochondrial mechanism of action and therapeutic effects. [PDF]
Biomed PharmacotherSabbah HN +8 more
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Distinct effects of tafazzin deletion in differentiated and undifferentiated mitochondria [PDF]
Mitochondrion, 2009 Tafazzin is a conserved mitochondrial protein that is required to maintain normal content and composition of cardiolipin. We used electron tomography to investigate the effect of tafazzin deletion on mitochondrial structure and found that cellular differentiation plays a crucial role in the manifestation of abnormalities. This conclusion was reached by
Riekelt H Houtkooper +2 more
exaly +4 more sources