Results 121 to 130 of about 2,318 (154)
3D bioprinted myocardium patches for rare Barth syndrome: TAZ mutation correction in cardioblasts. [PDF]
Tahir A, Imtiaz E, Mahato RK.
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Plasmalogens as biomarkers and therapeutic targets. [PDF]
Curran CS, Remaley AT, Torabi-Parizi P.
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Mitochondrial membrane lipids in the regulation of bioenergetic flux. [PDF]
Decker ST, Funai K.
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Cardiolipin preserves T<sub>reg</sub> metabolic fitness and immune homeostasis in the gut. [PDF]
Regina A +31 more
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Mitochondrial cristae remodeling: Mechanisms, functions, and pathology. [PDF]
Yu J +7 more
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Mitochondrial cardiolipin remodeling facilitates efficient myoblast differentiation. [PDF]
Ohba Y, Fujiwara CA, Arita M.
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Beyond Powerhouses: Roles of Mitochondria, from Development to Therapeutic Potential. [PDF]
Pereira SP, Tavares LC.
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Cardiolipin remodeling and the function of tafazzin
Biochimica Et Biophysica Acta - Molecular and Cell Biology of Lipids, 2013Cardiolipin, the specific phospholipid of mitochondria, is involved in the biogenesis, the dynamics, and the supramolecular organization of mitochondrial membranes. Cardiolipin acquires a characteristic composition of fatty acids by post-synthetic remodeling, a process that is crucial for cardiolipin homeostasis and function.
Michael Schlame
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Distinct effects of tafazzin deletion in differentiated and undifferentiated mitochondria [PDF]
Tafazzin is a conserved mitochondrial protein that is required to maintain normal content and composition of cardiolipin. We used electron tomography to investigate the effect of tafazzin deletion on mitochondrial structure and found that cellular differentiation plays a crucial role in the manifestation of abnormalities. This conclusion was reached by
Riekelt H Houtkooper +2 more
exaly +4 more sources

