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Cell-Penetrating Peptide Enhances Tafazzin Gene Therapy in Mouse Model of Barth Syndrome [PDF]

International Journal of Molecular Sciences
Barth Syndrome (BTHS) is an early onset, lethal X-linked disorder caused by a mutation in tafazzin (TAFAZZIN), a mitochondrial acyltransferase that remodels monolysocardiolipin (MLCL) to mature cardiolipin (CL) and is essential for normal mitochondrial ...
Rahul Raghav, Robert M Blanton, Douglas Strathdee   +2 more
exaly   +4 more sources

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The lipid environment modulates cardiolipin and phospholipid constitution in wild type and tafazzin-deficient cells

Journal of Inherited Metabolic Disease, 2022
Yvonne Wohlfarter, Stephan Geley, Gregor Oemer   +2 more
exaly  

Tafazzin deficiency in mouse mesenchymal stem cells promote reprogramming of activated B lymphocytes toward immunosuppressive phenotypes

FASEB Journal, 2022
Ejlal Abu-El-Rub, Aaron J. Marshall, Grant M Hatch   +2 more
exaly  

Role of Tafazzin in Mitochondrial Function, Development and Disease

Journal of Developmental Biology, 2020
Simon J. Conway, Michael T Chin
exaly  

A Zebrafish Model of Human Barth Syndrome Reveals the Essential Role of Tafazzin in Cardiac Development and Function

Circulation Research, 2006
Zaza Khuchua, Arnold W Strauss
exaly  

The Mitochondrial Transacylase, Tafazzin, Regulates AML Stemness by Modulating Intracellular Levels of Phospholipids

Cell Stem Cell, 2019
Steven M. Claypool, Ken Stark, Veronique Voisin   +2 more
exaly  

Distinct effects of tafazzin deletion in differentiated and undifferentiated mitochondria

Mitochondrion, 2009
Riekelt H Houtkooper, Frédéric M Vaz, Michael Schlame   +2 more
exaly  

Cardiolipin remodeling and the function of tafazzin

Biochimica Et Biophysica Acta - Molecular and Cell Biology of Lipids, 2013
Michael Schlame
exaly  

Mechanism for Remodeling of the Acyl Chain Composition of Cardiolipin Catalyzed by Saccharomyces cerevisiae Tafazzin*

Journal of Biological Chemistry, 2016
Masahide Oku, Hideto Miyoshi, Yasuyoshi Sakai   +2 more
exaly  

Suppression of Tafazzin promotes thyroid cancer apoptosis via activating the JNK signaling pathway and enhancing INF2-mediated mitochondrial fission

Journal of Cellular Physiology, 2019
Ziwen Liu, Hongwei Yuan, Mengwei Wu
exaly