Plain language summary of the Update of the risk assessment on dioxins and dioxin-like PCBs in feed and food. [PDF]
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The aryl hydrocarbon receptor: structure, signaling, physiology and pathology. [PDF]
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Reimagining AHR in Cancer: From Environmental Sensor to Novel Immunomodulatory Therapeutic Target. [PDF]
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Endocrine Disruptors and Breast Cancer: A Comprehensive Review. [PDF]
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Transcriptome study exploring the mechanisms linking pollutants and breast cancer aggressiveness. [PDF]
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Mixture Effects of Metals, PCBs, Dioxins, and Furans on Liver Function. [PDF]
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Evaluating the Effects of Neburon Exposure on Ovarian Folliculogenesis Using Zebrafish and Mouse Granulosa Cell Line. [PDF]
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To investigate the relationship between vitamin A status and 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) lethality, the influence of TCDD on tissue and serum vitamin A levels was determined in the most TCDD-susceptible (Long-Evans) and the most TCDD-resistant (Han/Wistar) rat strains.
Helen Håkansson, R Pohjanvirta
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Secretion of TCDD in milk and cream following the feeding of TCDD to lactating dairy cows
Bulletin of Environmental Contamination and Toxicology, 1982This study has shown that the level of TCDD secreted into milk and cream are such as to suggest that the residue found is proportional to the TCDD diet level and to the fat content of the milk or cream. After TCDD is removed from the diet the amount of TCDD secreted into milk decreases with time having a half-life of 41 days.
D J, Jensen, R A, Hummel
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Fate and transport of 1278-TCDD, 1378-TCDD, and 1478-TCDD in soil–water systems
Science of The Total Environment, 2006The most toxic dioxin is 2,3,7,8-tetrachlorodibenzo-p-dioxin (2378-TCDD), and obtaining comprehensive experimental data for this compound is challenging. However, several nontoxic isomers of 2378-TCDD exist, and can provide significant experimental evidence about this highly toxic dioxin.
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